pubmed-article:21415394 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:21415394 | lifeskim:mentions | umls-concept:C0035820 | lld:lifeskim |
pubmed-article:21415394 | lifeskim:mentions | umls-concept:C0014257 | lld:lifeskim |
pubmed-article:21415394 | lifeskim:mentions | umls-concept:C0006784 | lld:lifeskim |
pubmed-article:21415394 | lifeskim:mentions | umls-concept:C0529330 | lld:lifeskim |
pubmed-article:21415394 | lifeskim:mentions | umls-concept:C0277785 | lld:lifeskim |
pubmed-article:21415394 | lifeskim:mentions | umls-concept:C1514762 | lld:lifeskim |
pubmed-article:21415394 | lifeskim:mentions | umls-concept:C0205263 | lld:lifeskim |
pubmed-article:21415394 | lifeskim:mentions | umls-concept:C0679622 | lld:lifeskim |
pubmed-article:21415394 | lifeskim:mentions | umls-concept:C1879547 | lld:lifeskim |
pubmed-article:21415394 | lifeskim:mentions | umls-concept:C0205314 | lld:lifeskim |
pubmed-article:21415394 | pubmed:issue | 9 | lld:pubmed |
pubmed-article:21415394 | pubmed:dateCreated | 2011-4-29 | lld:pubmed |
pubmed-article:21415394 | pubmed:abstractText | The cytosolic protease calpain has been recently implicated in the vascular remodeling of angiotensin II (Ang II) type 1 receptor (AT(1)R) signaling. The role of Ang II/AT(1)R/calpain signaling on endothelial function, an important and early determinant of vascular pathology, remains though totally unknown. Accordingly, we investigated the role of calpain in the endothelial dysfunction of Ang II. | lld:pubmed |
pubmed-article:21415394 | pubmed:grant | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:21415394 | pubmed:grant | http://linkedlifedata.com/r... | lld:pubmed |
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pubmed-article:21415394 | pubmed:language | eng | lld:pubmed |
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pubmed-article:21415394 | pubmed:citationSubset | IM | lld:pubmed |
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pubmed-article:21415394 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:21415394 | pubmed:month | Apr | lld:pubmed |
pubmed-article:21415394 | pubmed:issn | 1524-4571 | lld:pubmed |
pubmed-article:21415394 | pubmed:author | pubmed-author:ScaliaRosario... | lld:pubmed |
pubmed-article:21415394 | pubmed:author | pubmed-author:GongYulanY | lld:pubmed |
pubmed-article:21415394 | pubmed:author | pubmed-author:BerzinsBrettB | lld:pubmed |
pubmed-article:21415394 | pubmed:author | pubmed-author:MishraGouravG | lld:pubmed |
pubmed-article:21415394 | pubmed:author | pubmed-author:FeatherDaniel... | lld:pubmed |
pubmed-article:21415394 | pubmed:author | pubmed-author:FreundBrinB | lld:pubmed |
pubmed-article:21415394 | pubmed:author | pubmed-author:LandesbergGav... | lld:pubmed |
pubmed-article:21415394 | pubmed:issnType | Electronic | lld:pubmed |
pubmed-article:21415394 | pubmed:day | 29 | lld:pubmed |
pubmed-article:21415394 | pubmed:volume | 108 | lld:pubmed |
pubmed-article:21415394 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:21415394 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:21415394 | pubmed:pagination | 1102-11 | lld:pubmed |
pubmed-article:21415394 | pubmed:dateRevised | 2011-8-1 | lld:pubmed |
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pubmed-article:21415394 | pubmed:meshHeading | pubmed-meshheading:21415394... | lld:pubmed |
pubmed-article:21415394 | pubmed:meshHeading | pubmed-meshheading:21415394... | lld:pubmed |
pubmed-article:21415394 | pubmed:meshHeading | pubmed-meshheading:21415394... | lld:pubmed |
pubmed-article:21415394 | pubmed:meshHeading | pubmed-meshheading:21415394... | lld:pubmed |
pubmed-article:21415394 | pubmed:year | 2011 | lld:pubmed |
pubmed-article:21415394 | pubmed:articleTitle | A novel role for calpain in the endothelial dysfunction induced by activation of angiotensin II type 1 receptor signaling. | lld:pubmed |
pubmed-article:21415394 | pubmed:affiliation | Department of Physiology, and The Cardiovascular Research Center, Temple University, Philadelphia, PA 19140, USA. rscalia@temple.edu | lld:pubmed |
pubmed-article:21415394 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:21415394 | pubmed:publicationType | Research Support, N.I.H., Extramural | lld:pubmed |
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