pubmed-article:21383962 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:21383962 | lifeskim:mentions | umls-concept:C0035647 | lld:lifeskim |
pubmed-article:21383962 | lifeskim:mentions | umls-concept:C1302401 | lld:lifeskim |
pubmed-article:21383962 | lifeskim:mentions | umls-concept:C0016030 | lld:lifeskim |
pubmed-article:21383962 | lifeskim:mentions | umls-concept:C0018270 | lld:lifeskim |
pubmed-article:21383962 | lifeskim:mentions | umls-concept:C0332281 | lld:lifeskim |
pubmed-article:21383962 | lifeskim:mentions | umls-concept:C0205217 | lld:lifeskim |
pubmed-article:21383962 | lifeskim:mentions | umls-concept:C0175630 | lld:lifeskim |
pubmed-article:21383962 | pubmed:dateCreated | 2011-3-8 | lld:pubmed |
pubmed-article:21383962 | pubmed:abstractText | Fibroblast growth factor-23 (FGF-23) is a phosphaturic peptide and a key component of an endocrine feedback loop along with the hormonal vitamin D metabolite 1,25(OH)(2)D. Vitamin D has been shown to be inversely related to colorectal neoplasia; therefore, we hypothesized that the effect of FGF-23 on vitamin D metabolite concentrations could have implications for the risk of colorectal neoplasia. | lld:pubmed |
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pubmed-article:21383962 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:21383962 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
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pubmed-article:21383962 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:21383962 | pubmed:language | eng | lld:pubmed |
pubmed-article:21383962 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:21383962 | pubmed:status | PubMed-not-MEDLINE | lld:pubmed |
pubmed-article:21383962 | pubmed:issn | 1477-3163 | lld:pubmed |
pubmed-article:21383962 | pubmed:author | pubmed-author:LancePeterP | lld:pubmed |
pubmed-article:21383962 | pubmed:author | pubmed-author:MartinezMaria... | lld:pubmed |
pubmed-article:21383962 | pubmed:author | pubmed-author:MayMelissaM | lld:pubmed |
pubmed-article:21383962 | pubmed:author | pubmed-author:BuckmeierJuli... | lld:pubmed |
pubmed-article:21383962 | pubmed:author | pubmed-author:JacobsElizabe... | lld:pubmed |
pubmed-article:21383962 | pubmed:author | pubmed-author:JurutkaPeterP | lld:pubmed |
pubmed-article:21383962 | pubmed:issnType | Electronic | lld:pubmed |
pubmed-article:21383962 | pubmed:volume | 10 | lld:pubmed |
pubmed-article:21383962 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:21383962 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:21383962 | pubmed:pagination | 3 | lld:pubmed |
pubmed-article:21383962 | pubmed:dateRevised | 2011-7-26 | lld:pubmed |
pubmed-article:21383962 | pubmed:year | 2011 | lld:pubmed |
pubmed-article:21383962 | pubmed:articleTitle | Circulating fibroblast growth factor-23 is associated with increased risk for metachronous colorectal adenoma. | lld:pubmed |
pubmed-article:21383962 | pubmed:affiliation | Epidemiology and Biostatistics, Mel and Enid Zuckerman College of Public Health, Arizona Cancer Center, University of Arizona, P.O. Box 245024, Tucson, AZ 85724-5024, USA. | lld:pubmed |
pubmed-article:21383962 | pubmed:publicationType | Journal Article | lld:pubmed |