| pubmed-article:21320717 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:21320717 | lifeskim:mentions | umls-concept:C0020517 | lld:lifeskim |
| pubmed-article:21320717 | lifeskim:mentions | umls-concept:C0312740 | lld:lifeskim |
| pubmed-article:21320717 | lifeskim:mentions | umls-concept:C0034790 | lld:lifeskim |
| pubmed-article:21320717 | lifeskim:mentions | umls-concept:C0118111 | lld:lifeskim |
| pubmed-article:21320717 | lifeskim:mentions | umls-concept:C0205216 | lld:lifeskim |
| pubmed-article:21320717 | lifeskim:mentions | umls-concept:C1514762 | lld:lifeskim |
| pubmed-article:21320717 | lifeskim:mentions | umls-concept:C1424244 | lld:lifeskim |
| pubmed-article:21320717 | lifeskim:mentions | umls-concept:C0220905 | lld:lifeskim |
| pubmed-article:21320717 | lifeskim:mentions | umls-concept:C1524003 | lld:lifeskim |
| pubmed-article:21320717 | pubmed:issue | 5 | lld:pubmed |
| pubmed-article:21320717 | pubmed:dateCreated | 2011-5-2 | lld:pubmed |
| pubmed-article:21320717 | pubmed:abstractText | Allergy, the most common disease of immune dysregulation, has a substantial genetic component that is poorly understood. Although complete disruption of T-cell receptor (TCR) signaling causes profound immunodeficiency, little is known about the consequences of inherited genetic variants that cause partial quantitative decreases in particular TCR-signaling pathways, despite their potential to dysregulate immune responses and cause immunopathology. | lld:pubmed |
| pubmed-article:21320717 | pubmed:language | eng | lld:pubmed |
| pubmed-article:21320717 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:21320717 | pubmed:citationSubset | AIM | lld:pubmed |
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| pubmed-article:21320717 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:21320717 | pubmed:month | May | lld:pubmed |
| pubmed-article:21320717 | pubmed:issn | 1097-6825 | lld:pubmed |
| pubmed-article:21320717 | pubmed:author | pubmed-author:GoodnowChrist... | lld:pubmed |
| pubmed-article:21320717 | pubmed:author | pubmed-author:BertramEdward... | lld:pubmed |
| pubmed-article:21320717 | pubmed:author | pubmed-author:CookMatthew... | lld:pubmed |
| pubmed-article:21320717 | pubmed:author | pubmed-author:ListonAdrianA | lld:pubmed |
| pubmed-article:21320717 | pubmed:author | pubmed-author:SOGADD | lld:pubmed |
| pubmed-article:21320717 | pubmed:author | pubmed-author:AltinJohn AJA | lld:pubmed |
| pubmed-article:21320717 | pubmed:copyrightInfo | Copyright © 2011 American Academy of Allergy, Asthma & Immunology. Published by Mosby, Inc. All rights reserved. | lld:pubmed |
| pubmed-article:21320717 | pubmed:issnType | Electronic | lld:pubmed |
| pubmed-article:21320717 | pubmed:volume | 127 | lld:pubmed |
| pubmed-article:21320717 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:21320717 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:21320717 | pubmed:pagination | 1277-85.e5 | lld:pubmed |
| pubmed-article:21320717 | pubmed:dateRevised | 2011-10-10 | lld:pubmed |
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| pubmed-article:21320717 | pubmed:year | 2011 | lld:pubmed |
| pubmed-article:21320717 | pubmed:articleTitle | Decreased T-cell receptor signaling through CARD11 differentially compromises forkhead box protein 3-positive regulatory versus T(H)2 effector cells to cause allergy. | lld:pubmed |
| pubmed-article:21320717 | pubmed:affiliation | Department of Immunology, John Curtin School of Medical Research, Australian National University, Canberra, Australia. | lld:pubmed |
| pubmed-article:21320717 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:21320717 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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