| pubmed-article:21276221 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:21276221 | lifeskim:mentions | umls-concept:C0006142 | lld:lifeskim |
| pubmed-article:21276221 | lifeskim:mentions | umls-concept:C0027627 | lld:lifeskim |
| pubmed-article:21276221 | lifeskim:mentions | umls-concept:C0033414 | lld:lifeskim |
| pubmed-article:21276221 | lifeskim:mentions | umls-concept:C2699153 | lld:lifeskim |
| pubmed-article:21276221 | lifeskim:mentions | umls-concept:C1269955 | lld:lifeskim |
| pubmed-article:21276221 | lifeskim:mentions | umls-concept:C0017262 | lld:lifeskim |
| pubmed-article:21276221 | lifeskim:mentions | umls-concept:C1306673 | lld:lifeskim |
| pubmed-article:21276221 | lifeskim:mentions | umls-concept:C1707520 | lld:lifeskim |
| pubmed-article:21276221 | lifeskim:mentions | umls-concept:C0205250 | lld:lifeskim |
| pubmed-article:21276221 | lifeskim:mentions | umls-concept:C0205179 | lld:lifeskim |
| pubmed-article:21276221 | lifeskim:mentions | umls-concept:C2911684 | lld:lifeskim |
| pubmed-article:21276221 | lifeskim:mentions | umls-concept:C0185117 | lld:lifeskim |
| pubmed-article:21276221 | pubmed:issue | 1 | lld:pubmed |
| pubmed-article:21276221 | pubmed:dateCreated | 2011-2-14 | lld:pubmed |
| pubmed-article:21276221 | pubmed:abstractText | B-lymphoma Moloney murine leukemia virus insertion region-1 (Bmi-1) acts as an oncogene in various tumors, and its overexpression correlates with a poor outcome in several human cancers. Ectopic expression of Bmi-1 can induce epithelial-mesenchymal transition (EMT) and enhance the motility and invasiveness of human nasopharyngeal epithelial cells (NPECs), whereas silencing endogenous Bmi-1 expression can reverse EMT and reduce the metastatic potential of nasopharyngeal cancer cells (NPCs). Mouse xenograft studies indicate that coexpression of Bmi-1 and H-Ras in breast cancer cells can induce an aggressive and metastatic phenotype with an unusual occurrence of brain metastasis; although, Bmi-1 overexpression did not result in oncogenic transformation of MCF-10A cells. However, the underlying molecular mechanism of Bmi-1-mediated progression and the metastasis of breast cancer are not fully elucidated at this time. | lld:pubmed |
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