pubmed-article:21270417 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:21270417 | lifeskim:mentions | umls-concept:C0039082 | lld:lifeskim |
pubmed-article:21270417 | lifeskim:mentions | umls-concept:C0242656 | lld:lifeskim |
pubmed-article:21270417 | lifeskim:mentions | umls-concept:C0205409 | lld:lifeskim |
pubmed-article:21270417 | lifeskim:mentions | umls-concept:C0231221 | lld:lifeskim |
pubmed-article:21270417 | lifeskim:mentions | umls-concept:C0241224 | lld:lifeskim |
pubmed-article:21270417 | lifeskim:mentions | umls-concept:C0681842 | lld:lifeskim |
pubmed-article:21270417 | pubmed:issue | 8 | lld:pubmed |
pubmed-article:21270417 | pubmed:dateCreated | 2011-2-22 | lld:pubmed |
pubmed-article:21270417 | pubmed:abstractText | Technological advancements in neuroimaging and the increased use of these diagnostic modalities are responsible for the discovery of incidentally identified anomalies within the CNS. In addition to the identification of unanticipated brain MRI abnormalities suggestive of demyelinating disease in patients undergoing neuroimaging for a medical reason other than evaluation for multiple sclerosis (MS), asymptomatic spinal cord lesions are periodically identified. | lld:pubmed |
pubmed-article:21270417 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:21270417 | pubmed:language | eng | lld:pubmed |
pubmed-article:21270417 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:21270417 | pubmed:citationSubset | AIM | lld:pubmed |
pubmed-article:21270417 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:21270417 | pubmed:month | Feb | lld:pubmed |
pubmed-article:21270417 | pubmed:issn | 1526-632X | lld:pubmed |
pubmed-article:21270417 | pubmed:author | pubmed-author:GoodinD SDS | lld:pubmed |
pubmed-article:21270417 | pubmed:author | pubmed-author:PelletierDD | lld:pubmed |
pubmed-article:21270417 | pubmed:author | pubmed-author:WaubantEE | lld:pubmed |
pubmed-article:21270417 | pubmed:author | pubmed-author:CreeB A CBA | lld:pubmed |
pubmed-article:21270417 | pubmed:author | pubmed-author:OkudaD TDT | lld:pubmed |
pubmed-article:21270417 | pubmed:author | pubmed-author:MowryE MEM | lld:pubmed |
pubmed-article:21270417 | pubmed:author | pubmed-author:CrabtreeE CEC | lld:pubmed |
pubmed-article:21270417 | pubmed:issnType | Electronic | lld:pubmed |
pubmed-article:21270417 | pubmed:day | 22 | lld:pubmed |
pubmed-article:21270417 | pubmed:volume | 76 | lld:pubmed |
pubmed-article:21270417 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:21270417 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:21270417 | pubmed:pagination | 686-92 | lld:pubmed |
pubmed-article:21270417 | pubmed:meshHeading | pubmed-meshheading:21270417... | lld:pubmed |
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pubmed-article:21270417 | pubmed:meshHeading | pubmed-meshheading:21270417... | lld:pubmed |
pubmed-article:21270417 | pubmed:year | 2011 | lld:pubmed |
pubmed-article:21270417 | pubmed:articleTitle | Asymptomatic spinal cord lesions predict disease progression in radiologically isolated syndrome. | lld:pubmed |
pubmed-article:21270417 | pubmed:affiliation | Department of Neurology, UCSF Multiple Sclerosis Center, University of California, San Francisco, San Francisco, USA. darin.okuda@chw.edu | lld:pubmed |
pubmed-article:21270417 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:21270417 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
pubmed-article:21270417 | pubmed:publicationType | Research Support, N.I.H., Extramural | lld:pubmed |
http://linkedlifedata.com/r... | pubmed:referesTo | pubmed-article:21270417 | lld:pubmed |