| pubmed-article:2126748 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:2126748 | lifeskim:mentions | umls-concept:C0006644 | lld:lifeskim |
| pubmed-article:2126748 | lifeskim:mentions | umls-concept:C0028351 | lld:lifeskim |
| pubmed-article:2126748 | lifeskim:mentions | umls-concept:C1278872 | lld:lifeskim |
| pubmed-article:2126748 | lifeskim:mentions | umls-concept:C0459521 | lld:lifeskim |
| pubmed-article:2126748 | lifeskim:mentions | umls-concept:C0033414 | lld:lifeskim |
| pubmed-article:2126748 | lifeskim:mentions | umls-concept:C0030685 | lld:lifeskim |
| pubmed-article:2126748 | lifeskim:mentions | umls-concept:C0205100 | lld:lifeskim |
| pubmed-article:2126748 | lifeskim:mentions | umls-concept:C0037633 | lld:lifeskim |
| pubmed-article:2126748 | lifeskim:mentions | umls-concept:C0680255 | lld:lifeskim |
| pubmed-article:2126748 | lifeskim:mentions | umls-concept:C0205263 | lld:lifeskim |
| pubmed-article:2126748 | lifeskim:mentions | umls-concept:C0391871 | lld:lifeskim |
| pubmed-article:2126748 | lifeskim:mentions | umls-concept:C1283071 | lld:lifeskim |
| pubmed-article:2126748 | lifeskim:mentions | umls-concept:C1963578 | lld:lifeskim |
| pubmed-article:2126748 | lifeskim:mentions | umls-concept:C1382100 | lld:lifeskim |
| pubmed-article:2126748 | pubmed:issue | 9 | lld:pubmed |
| pubmed-article:2126748 | pubmed:dateCreated | 1991-3-26 | lld:pubmed |
| pubmed-article:2126748 | pubmed:abstractText | The transmitter releasing action of caffeine was studied in the absence of extracellular Ca2+ from the peripheral sympathetic nerves of the rabbit main pulmonary artery. Caffeine (10 mM) increased the release of [3H]-noradrenaline moderately, but not significantly in Ca2(+)-free (+1 mM EGTA) Krebs solution. When peripheral nerve endings/varicosities were depolarized by elevating extracellular K+ to 47.2 mM and 70.8 mM in Ca2(+)-free solution, the transmitter releasing effect of 10 mM caffeine became significant. Ca2+ removal itself transiently increased the [3H]-noradrenaline outflow. In the individual experiments the amount of the caffeine evoked transmitter release at 47.2 mM and 70.8 mM K(+)-depolarization was inversely correlated to the release evoked by Ca2(+)-removal. Our results suggest that caffeine-sensitive calcium stores are present in peripheral nerve terminals of rabbit pulmonary artery, and part of the caffeine sensitive calcium stores may discharge during Ca2(+)-removal from the extracellular solution. | lld:pubmed |
| pubmed-article:2126748 | pubmed:language | eng | lld:pubmed |
| pubmed-article:2126748 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:2126748 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:2126748 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:2126748 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:2126748 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:2126748 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:2126748 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:2126748 | pubmed:month | Oct | lld:pubmed |
| pubmed-article:2126748 | pubmed:issn | 0143-4160 | lld:pubmed |
| pubmed-article:2126748 | pubmed:author | pubmed-author:MagyarKK | lld:pubmed |
| pubmed-article:2126748 | pubmed:author | pubmed-author:TörökT LTL | lld:pubmed |
| pubmed-article:2126748 | pubmed:author | pubmed-author:TóthP TPT | lld:pubmed |
| pubmed-article:2126748 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:2126748 | pubmed:volume | 11 | lld:pubmed |
| pubmed-article:2126748 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:2126748 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:2126748 | pubmed:pagination | 557-63 | lld:pubmed |
| pubmed-article:2126748 | pubmed:dateRevised | 2003-11-14 | lld:pubmed |
| pubmed-article:2126748 | pubmed:meshHeading | pubmed-meshheading:2126748-... | lld:pubmed |
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| pubmed-article:2126748 | pubmed:meshHeading | pubmed-meshheading:2126748-... | lld:pubmed |
| pubmed-article:2126748 | pubmed:meshHeading | pubmed-meshheading:2126748-... | lld:pubmed |
| pubmed-article:2126748 | pubmed:meshHeading | pubmed-meshheading:2126748-... | lld:pubmed |
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| pubmed-article:2126748 | pubmed:meshHeading | pubmed-meshheading:2126748-... | lld:pubmed |
| pubmed-article:2126748 | pubmed:meshHeading | pubmed-meshheading:2126748-... | lld:pubmed |
| pubmed-article:2126748 | pubmed:meshHeading | pubmed-meshheading:2126748-... | lld:pubmed |
| pubmed-article:2126748 | pubmed:meshHeading | pubmed-meshheading:2126748-... | lld:pubmed |
| pubmed-article:2126748 | pubmed:year | 1990 | lld:pubmed |
| pubmed-article:2126748 | pubmed:articleTitle | Depolarization promotes caffeine induced [3H]-noradrenaline release in calcium-free solution from peripheral sympathetic nerves. | lld:pubmed |
| pubmed-article:2126748 | pubmed:affiliation | Department of Pharmacodynamics, Semmelweis University of Medicine, Budapest, Hungary. | lld:pubmed |
| pubmed-article:2126748 | pubmed:publicationType | Journal Article | lld:pubmed |
| http://linkedlifedata.com/r... | pubmed:referesTo | pubmed-article:2126748 | lld:pubmed |
