| pubmed-article:212554 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:212554 | lifeskim:mentions | umls-concept:C0006675 | lld:lifeskim |
| pubmed-article:212554 | lifeskim:mentions | umls-concept:C0035820 | lld:lifeskim |
| pubmed-article:212554 | lifeskim:mentions | umls-concept:C1882715 | lld:lifeskim |
| pubmed-article:212554 | lifeskim:mentions | umls-concept:C0597357 | lld:lifeskim |
| pubmed-article:212554 | lifeskim:mentions | umls-concept:C0032821 | lld:lifeskim |
| pubmed-article:212554 | lifeskim:mentions | umls-concept:C0031164 | lld:lifeskim |
| pubmed-article:212554 | lifeskim:mentions | umls-concept:C2587213 | lld:lifeskim |
| pubmed-article:212554 | lifeskim:mentions | umls-concept:C0127400 | lld:lifeskim |
| pubmed-article:212554 | pubmed:dateCreated | 1978-12-27 | lld:pubmed |
| pubmed-article:212554 | pubmed:abstractText | 1. In the presence of extracellular Ca, adrenaline stimulated a large increase in the rate of K (86Rb) release from rat lacrimal slices, followed by a lower, more sustained rate. 2. In the absence of extracellular Ca, adrenaline elicited only a transient release of 86Rb. 3. The artificial introduction of Ca into the cytosol by the ionophore A-23187 could also initiate the release of 86Rb. 4. In a zero-Ca medium, if either adrenaline or carbachol produced a transient release of 86Rb, the tissue could not respond to the other agonist with a transient release unless Ca was momentarily reintroduced to the medium. 5. If Ca was present in a limiting concentration, the Ca-dependent rate of 86Rb release elicited from a lacrimal slice exposed simultaneously to carbachol and adrenaline was not significantly different from the release seen with carbachol alone. 6. It is concluded that the agonist-induced release of K from the lacrimal gland consists of both a Ca-independent phase which is initiated by the release of a limited pool of Ca, and a Ca-dependent phase which is mediated by the influx of extracellular Ca. 7. It is also concluded that both alpha-adrenergic and muscarinic receptor occupation activate a common, post-receptor mechanism which may be responsible for both phases of K release. | lld:pubmed |
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| pubmed-article:212554 | pubmed:language | eng | lld:pubmed |
| pubmed-article:212554 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:212554 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:212554 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:212554 | pubmed:month | Aug | lld:pubmed |
| pubmed-article:212554 | pubmed:issn | 0022-3751 | lld:pubmed |
| pubmed-article:212554 | pubmed:author | pubmed-author:PutneyJ WJWJr | lld:pubmed |
| pubmed-article:212554 | pubmed:author | pubmed-author:ParodR JRJ | lld:pubmed |
| pubmed-article:212554 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:212554 | pubmed:volume | 281 | lld:pubmed |
| pubmed-article:212554 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:212554 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:212554 | pubmed:pagination | 371-81 | lld:pubmed |
| pubmed-article:212554 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
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| pubmed-article:212554 | pubmed:year | 1978 | lld:pubmed |
| pubmed-article:212554 | pubmed:articleTitle | The role of calcium in the receptor mediated control of potassium permeability in the rat lacrimal gland. | lld:pubmed |
| pubmed-article:212554 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:212554 | pubmed:publicationType | In Vitro | lld:pubmed |
| pubmed-article:212554 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
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