| pubmed-article:21254366 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:21254366 | lifeskim:mentions | umls-concept:C0018798 | lld:lifeskim |
| pubmed-article:21254366 | lifeskim:mentions | umls-concept:C1858460 | lld:lifeskim |
| pubmed-article:21254366 | lifeskim:mentions | umls-concept:C2613367 | lld:lifeskim |
| pubmed-article:21254366 | pubmed:issue | 2 | lld:pubmed |
| pubmed-article:21254366 | pubmed:dateCreated | 2011-2-14 | lld:pubmed |
| pubmed-article:21254366 | pubmed:abstractText | Congenital heart defects (CHDs) are among the most prevalent and serious of birth defects. Multiple maternal factors are thought to contribute to CHD development, including folate intake. Maternal DNA methylation, which is dependent on folate metabolism, may impact the risk of CHDs. Our study was designed to determine whether maternal long interspersed nucleotide elements-1 (LINE-1) DNA hypomethylation is associated with increased occurrence of non-syndromic CHDs and whether maternal folate-dependent metabolites are correlated with DNA methylation status. | lld:pubmed |
| pubmed-article:21254366 | pubmed:grant | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:21254366 | pubmed:grant | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:21254366 | pubmed:grant | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:21254366 | pubmed:language | eng | lld:pubmed |
| pubmed-article:21254366 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:21254366 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:21254366 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:21254366 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:21254366 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:21254366 | pubmed:month | Feb | lld:pubmed |
| pubmed-article:21254366 | pubmed:issn | 1542-0760 | lld:pubmed |
| pubmed-article:21254366 | pubmed:author | pubmed-author:JamesS JillSJ | lld:pubmed |
| pubmed-article:21254366 | pubmed:author | pubmed-author:HobbsCharlott... | lld:pubmed |
| pubmed-article:21254366 | pubmed:author | pubmed-author:ClevesMario... | lld:pubmed |
| pubmed-article:21254366 | pubmed:author | pubmed-author:ZhaoWeizhiW | lld:pubmed |
| pubmed-article:21254366 | pubmed:author | pubmed-author:MacLeodStewar... | lld:pubmed |
| pubmed-article:21254366 | pubmed:author | pubmed-author:ChowdhuryShim... | lld:pubmed |
| pubmed-article:21254366 | pubmed:copyrightInfo | Copyright © 2011 Wiley-Liss, Inc. | lld:pubmed |
| pubmed-article:21254366 | pubmed:issnType | Electronic | lld:pubmed |
| pubmed-article:21254366 | pubmed:volume | 91 | lld:pubmed |
| pubmed-article:21254366 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:21254366 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:21254366 | pubmed:pagination | 69-76 | lld:pubmed |
| pubmed-article:21254366 | pubmed:dateRevised | 2011-9-26 | lld:pubmed |
| pubmed-article:21254366 | pubmed:meshHeading | pubmed-meshheading:21254366... | lld:pubmed |
| pubmed-article:21254366 | pubmed:meshHeading | pubmed-meshheading:21254366... | lld:pubmed |
| pubmed-article:21254366 | pubmed:meshHeading | pubmed-meshheading:21254366... | lld:pubmed |
| pubmed-article:21254366 | pubmed:meshHeading | pubmed-meshheading:21254366... | lld:pubmed |
| pubmed-article:21254366 | pubmed:meshHeading | pubmed-meshheading:21254366... | lld:pubmed |
| pubmed-article:21254366 | pubmed:meshHeading | pubmed-meshheading:21254366... | lld:pubmed |
| pubmed-article:21254366 | pubmed:meshHeading | pubmed-meshheading:21254366... | lld:pubmed |
| pubmed-article:21254366 | pubmed:meshHeading | pubmed-meshheading:21254366... | lld:pubmed |
| pubmed-article:21254366 | pubmed:meshHeading | pubmed-meshheading:21254366... | lld:pubmed |
| pubmed-article:21254366 | pubmed:meshHeading | pubmed-meshheading:21254366... | lld:pubmed |
| pubmed-article:21254366 | pubmed:meshHeading | pubmed-meshheading:21254366... | lld:pubmed |
| pubmed-article:21254366 | pubmed:meshHeading | pubmed-meshheading:21254366... | lld:pubmed |
| pubmed-article:21254366 | pubmed:meshHeading | pubmed-meshheading:21254366... | lld:pubmed |
| pubmed-article:21254366 | pubmed:meshHeading | pubmed-meshheading:21254366... | lld:pubmed |
| pubmed-article:21254366 | pubmed:year | 2011 | lld:pubmed |
| pubmed-article:21254366 | pubmed:articleTitle | Maternal DNA hypomethylation and congenital heart defects. | lld:pubmed |
| pubmed-article:21254366 | pubmed:affiliation | Department of Pediatrics, College of Medicine, University of Arkansas for Medical Sciences, Arkansas Children's Hospital Research Institute, 13 Children's Way, Little Rock, AR 72202, USA. | lld:pubmed |
| pubmed-article:21254366 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:21254366 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
| pubmed-article:21254366 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
| pubmed-article:21254366 | pubmed:publicationType | Research Support, N.I.H., Extramural | lld:pubmed |
