pubmed-article:21223590 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:21223590 | lifeskim:mentions | umls-concept:C0026339 | lld:lifeskim |
pubmed-article:21223590 | lifeskim:mentions | umls-concept:C0025914 | lld:lifeskim |
pubmed-article:21223590 | lifeskim:mentions | umls-concept:C0026809 | lld:lifeskim |
pubmed-article:21223590 | lifeskim:mentions | umls-concept:C0026336 | lld:lifeskim |
pubmed-article:21223590 | lifeskim:mentions | umls-concept:C0017205 | lld:lifeskim |
pubmed-article:21223590 | lifeskim:mentions | umls-concept:C0449258 | lld:lifeskim |
pubmed-article:21223590 | lifeskim:mentions | umls-concept:C1956267 | lld:lifeskim |
pubmed-article:21223590 | lifeskim:mentions | umls-concept:C2348867 | lld:lifeskim |
pubmed-article:21223590 | pubmed:dateCreated | 2011-2-3 | lld:pubmed |
pubmed-article:21223590 | pubmed:abstractText | Gaucher disease is caused by defective glucocerebrosidase activity and the consequent accumulation of glucosylceramide. The pathogenic pathways resulting from lipid laden macrophages (Gaucher cells) in visceral organs and their abnormal functions are obscure. | lld:pubmed |
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pubmed-article:21223590 | pubmed:language | eng | lld:pubmed |
pubmed-article:21223590 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:21223590 | pubmed:citationSubset | IM | lld:pubmed |
pubmed-article:21223590 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:21223590 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:21223590 | pubmed:issn | 1471-2164 | lld:pubmed |
pubmed-article:21223590 | pubmed:author | pubmed-author:OsL OLO | lld:pubmed |
pubmed-article:21223590 | pubmed:author | pubmed-author:SunYingY | lld:pubmed |
pubmed-article:21223590 | pubmed:author | pubmed-author:SetchellKenne... | lld:pubmed |
pubmed-article:21223590 | pubmed:author | pubmed-author:AronowBruceB | lld:pubmed |