| pubmed-article:21182830 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:21182830 | lifeskim:mentions | umls-concept:C0035820 | lld:lifeskim |
| pubmed-article:21182830 | lifeskim:mentions | umls-concept:C0917798 | lld:lifeskim |
| pubmed-article:21182830 | lifeskim:mentions | umls-concept:C0521451 | lld:lifeskim |
| pubmed-article:21182830 | lifeskim:mentions | umls-concept:C0032824 | lld:lifeskim |
| pubmed-article:21182830 | lifeskim:mentions | umls-concept:C0205234 | lld:lifeskim |
| pubmed-article:21182830 | pubmed:dateCreated | 2011-2-7 | lld:pubmed |
| pubmed-article:21182830 | pubmed:abstractText | Ischemic postconditioning (IpostC) has been described in both heart and brain. The first aim of this study was to evaluate the effects of IpostC on brain infarct size and neurological function in the middle cerebral artery occlusion (MCAO) model. The second aim was to determine the involvement of the mitochondrial potassium ATP-dependent channel (mitoK(ATP)) opening and its capacity to improve mitochondrial dysfunction induced by ischemia-reperfusion. | lld:pubmed |
| pubmed-article:21182830 | pubmed:language | eng | lld:pubmed |
| pubmed-article:21182830 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:21182830 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:21182830 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:21182830 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:21182830 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:21182830 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:21182830 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:21182830 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:21182830 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:21182830 | pubmed:month | Feb | lld:pubmed |
| pubmed-article:21182830 | pubmed:issn | 1872-6240 | lld:pubmed |
| pubmed-article:21182830 | pubmed:author | pubmed-author:ValletBenoitB | lld:pubmed |
| pubmed-article:21182830 | pubmed:author | pubmed-author:LebuffeGilles... | lld:pubmed |
| pubmed-article:21182830 | pubmed:author | pubmed-author:BordetRégisR | lld:pubmed |
| pubmed-article:21182830 | pubmed:author | pubmed-author:RobinEmmanuel... | lld:pubmed |
| pubmed-article:21182830 | pubmed:author | pubmed-author:HassounSidi... | lld:pubmed |
| pubmed-article:21182830 | pubmed:author | pubmed-author:SimerabetMali... | lld:pubmed |
| pubmed-article:21182830 | pubmed:author | pubmed-author:AdamczykSebas... | lld:pubmed |
| pubmed-article:21182830 | pubmed:author | pubmed-author:TavernierBeno... | lld:pubmed |
| pubmed-article:21182830 | pubmed:copyrightInfo | Copyright © 2010 Elsevier B.V. All rights reserved. | lld:pubmed |
| pubmed-article:21182830 | pubmed:issnType | Electronic | lld:pubmed |
| pubmed-article:21182830 | pubmed:day | 23 | lld:pubmed |
| pubmed-article:21182830 | pubmed:volume | 1375 | lld:pubmed |
| pubmed-article:21182830 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:21182830 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:21182830 | pubmed:pagination | 137-46 | lld:pubmed |
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| pubmed-article:21182830 | pubmed:meshHeading | pubmed-meshheading:21182830... | lld:pubmed |
| pubmed-article:21182830 | pubmed:meshHeading | pubmed-meshheading:21182830... | lld:pubmed |
| pubmed-article:21182830 | pubmed:meshHeading | pubmed-meshheading:21182830... | lld:pubmed |
| pubmed-article:21182830 | pubmed:meshHeading | pubmed-meshheading:21182830... | lld:pubmed |
| pubmed-article:21182830 | pubmed:meshHeading | pubmed-meshheading:21182830... | lld:pubmed |
| pubmed-article:21182830 | pubmed:meshHeading | pubmed-meshheading:21182830... | lld:pubmed |
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| pubmed-article:21182830 | pubmed:meshHeading | pubmed-meshheading:21182830... | lld:pubmed |
| pubmed-article:21182830 | pubmed:year | 2011 | lld:pubmed |
| pubmed-article:21182830 | pubmed:articleTitle | Postconditioning in focal cerebral ischemia: role of the mitochondrial ATP-dependent potassium channel. | lld:pubmed |
| pubmed-article:21182830 | pubmed:affiliation | Department of Pharmacology, Lille Medical School, University of Lille 2, Lille, France. | lld:pubmed |
| pubmed-article:21182830 | pubmed:publicationType | Journal Article | lld:pubmed |
