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| pubmed-article:2117703 | lifeskim:mentions | umls-concept:C0332307 | lld:lifeskim |
| pubmed-article:2117703 | lifeskim:mentions | umls-concept:C1260875 | lld:lifeskim |
| pubmed-article:2117703 | lifeskim:mentions | umls-concept:C0019648 | lld:lifeskim |
| pubmed-article:2117703 | lifeskim:mentions | umls-concept:C1425774 | lld:lifeskim |
| pubmed-article:2117703 | lifeskim:mentions | umls-concept:C1706229 | lld:lifeskim |
| pubmed-article:2117703 | lifeskim:mentions | umls-concept:C1708726 | lld:lifeskim |
| pubmed-article:2117703 | lifeskim:mentions | umls-concept:C0162735 | lld:lifeskim |
| pubmed-article:2117703 | lifeskim:mentions | umls-concept:C1539104 | lld:lifeskim |
| pubmed-article:2117703 | pubmed:issue | 9 | lld:pubmed |
| pubmed-article:2117703 | pubmed:dateCreated | 1990-9-26 | lld:pubmed |
| pubmed-article:2117703 | pubmed:abstractText | The N-terminal serine and four conserved lysine residues near the N-terminus of yeast histone H4 are acetylated. We found that a mutation that changed the fourth lysine to alanine resulted in specific derepression of the silent mating type locus HML, while mutations that altered the N-terminal serine or the first three lysines had only minor phenotypic effects. Our results support an active role for histone H4 in the silencing of gene expression at this locus. | lld:pubmed |
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| pubmed-article:2117703 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:2117703 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:2117703 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:2117703 | pubmed:month | Sep | lld:pubmed |
| pubmed-article:2117703 | pubmed:issn | 0270-7306 | lld:pubmed |
| pubmed-article:2117703 | pubmed:author | pubmed-author:SzostakJ WJW | lld:pubmed |
| pubmed-article:2117703 | pubmed:author | pubmed-author:ParkE CEC | lld:pubmed |
| pubmed-article:2117703 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:2117703 | pubmed:volume | 10 | lld:pubmed |
| pubmed-article:2117703 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:2117703 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:2117703 | pubmed:pagination | 4932-4 | lld:pubmed |
| pubmed-article:2117703 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
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| pubmed-article:2117703 | pubmed:year | 1990 | lld:pubmed |
| pubmed-article:2117703 | pubmed:articleTitle | Point mutations in the yeast histone H4 gene prevent silencing of the silent mating type locus HML. | lld:pubmed |
| pubmed-article:2117703 | pubmed:affiliation | Department of Molecular Biology, Massachusetts General Hospital, Boston 02114. | lld:pubmed |
| pubmed-article:2117703 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:2117703 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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