pubmed-article:2117300 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:2117300 | lifeskim:mentions | umls-concept:C0025952 | lld:lifeskim |
pubmed-article:2117300 | lifeskim:mentions | umls-concept:C0014507 | lld:lifeskim |
pubmed-article:2117300 | lifeskim:mentions | umls-concept:C0085494 | lld:lifeskim |
pubmed-article:2117300 | lifeskim:mentions | umls-concept:C0017436 | lld:lifeskim |
pubmed-article:2117300 | lifeskim:mentions | umls-concept:C0314603 | lld:lifeskim |
pubmed-article:2117300 | lifeskim:mentions | umls-concept:C0683598 | lld:lifeskim |
pubmed-article:2117300 | lifeskim:mentions | umls-concept:C1527178 | lld:lifeskim |
pubmed-article:2117300 | pubmed:issue | 4 | lld:pubmed |
pubmed-article:2117300 | pubmed:dateCreated | 1990-9-17 | lld:pubmed |
pubmed-article:2117300 | pubmed:abstractText | Antibiotic resistance is an ever-increasing problem in enterococci. These bacteria are remarkable in their ability to acquire and disseminate antibiotic resistance genes by a variety of routes. Since first described in 1979, high-level resistance to gentamicin (MIC, greater than 2,000 micrograms/mL) has spread worldwide and has been responsible for serious infections. Resistance is plasmid-mediated and due to aminoglycoside-modifying enzymes. High-level gentamicin resistance indicates that there will be no synergistic bactericidal activity with penicillin-gentamicin combinations. The epidemiology of nosocomial enterococcal infections is remarkably similar to that of nosocomial infections caused by methicillin-resistant staphylococci and by multidrug-resistant gram-negative bacilli. The most likely way these resistant bacteria are spread among hospital patients is via transient carriage on the hands of hospital personnel. Patient-to-patient and interhospital transmission of strains has been reported recently. However, clonal dissemination is not the cause of the increased frequency of resistant strains, since gentamicin resistance appears in a variety of different conjugative and nonconjugative plasmids in Enterococcus. | lld:pubmed |
pubmed-article:2117300 | pubmed:language | eng | lld:pubmed |
pubmed-article:2117300 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:2117300 | pubmed:citationSubset | IM | lld:pubmed |
pubmed-article:2117300 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:2117300 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:2117300 | pubmed:issn | 0162-0886 | lld:pubmed |
pubmed-article:2117300 | pubmed:author | pubmed-author:PattersonJ... | lld:pubmed |
pubmed-article:2117300 | pubmed:author | pubmed-author:ZervosM JMJ | lld:pubmed |
pubmed-article:2117300 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:2117300 | pubmed:volume | 12 | lld:pubmed |
pubmed-article:2117300 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:2117300 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:2117300 | pubmed:pagination | 644-52 | lld:pubmed |
pubmed-article:2117300 | pubmed:dateRevised | 2006-11-15 | lld:pubmed |
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pubmed-article:2117300 | pubmed:meshHeading | pubmed-meshheading:2117300-... | lld:pubmed |
pubmed-article:2117300 | pubmed:articleTitle | High-level gentamicin resistance in Enterococcus: microbiology, genetic basis, and epidemiology. | lld:pubmed |
pubmed-article:2117300 | pubmed:affiliation | Department of Medicine and Laboratory Medicine, Yale University School of Medicine, New Haven, Connecticut. | lld:pubmed |
pubmed-article:2117300 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:2117300 | pubmed:publicationType | Review | lld:pubmed |
pubmed-article:2117300 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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