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pubmed-article:21049284pubmed:abstractTextPulmonary vasodilators in general and prostacyclin therapy in particular, have markedly improved the outcome of patients with pulmonary arterial hypertension (PAH). As endothelial dysfunction is a key feature of PAH, and as endothelial progenitor cells (EPC) may contribute to vascular repair in PAH, we suspected that prostacyclin therapy might enhance EPC numbers and functions. In the present study, objectives were to determine whether EPC may contribute to vasodilator treatment efficacy in PAH.lld:pubmed
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pubmed-article:21049284pubmed:authorpubmed-author:LévyMarilyneMlld:pubmed
pubmed-article:21049284pubmed:authorpubmed-author:GaussemPascal...lld:pubmed
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pubmed-article:21049284pubmed:authorpubmed-author:SmadjaDavid...lld:pubmed
pubmed-article:21049284pubmed:authorpubmed-author:d'AudigierClé...lld:pubmed
pubmed-article:21049284pubmed:authorpubmed-author:MaugeLaetitia...lld:pubmed
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pubmed-article:21049284pubmed:dateRevised2011-11-17lld:pubmed
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pubmed-article:21049284pubmed:articleTitleTreprostinil increases the number and angiogenic potential of endothelial progenitor cells in children with pulmonary hypertension.lld:pubmed
pubmed-article:21049284pubmed:affiliationUniversité Paris Descartes, Paris, France. david.smadja@egp.aphp.frlld:pubmed
pubmed-article:21049284pubmed:publicationTypeJournal Articlelld:pubmed
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