| pubmed-article:21045728 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:21045728 | lifeskim:mentions | umls-concept:C0020538 | lld:lifeskim |
| pubmed-article:21045728 | lifeskim:mentions | umls-concept:C1522318 | lld:lifeskim |
| pubmed-article:21045728 | lifeskim:mentions | umls-concept:C0021186 | lld:lifeskim |
| pubmed-article:21045728 | lifeskim:mentions | umls-concept:C0136123 | lld:lifeskim |
| pubmed-article:21045728 | lifeskim:mentions | umls-concept:C0332161 | lld:lifeskim |
| pubmed-article:21045728 | lifeskim:mentions | umls-concept:C1555029 | lld:lifeskim |
| pubmed-article:21045728 | lifeskim:mentions | umls-concept:C0806140 | lld:lifeskim |
| pubmed-article:21045728 | pubmed:issue | 2 | lld:pubmed |
| pubmed-article:21045728 | pubmed:dateCreated | 2010-12-30 | lld:pubmed |
| pubmed-article:21045728 | pubmed:abstractText | Patients and animal models of arterial hypertension are characterized by structural and functional abnormalities of the coronary microcirculation. Using a translational approach, we ascertained whether antihypertensive treatment can reverse microvascular remodelling and improve myocardial perfusion. | lld:pubmed |
| pubmed-article:21045728 | pubmed:language | eng | lld:pubmed |
| pubmed-article:21045728 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:21045728 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:21045728 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:21045728 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:21045728 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:21045728 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:21045728 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:21045728 | pubmed:month | Feb | lld:pubmed |
| pubmed-article:21045728 | pubmed:issn | 1473-5598 | lld:pubmed |
| pubmed-article:21045728 | pubmed:author | pubmed-author:NegliaDaniloD | lld:pubmed |
| pubmed-article:21045728 | pubmed:author | pubmed-author:CamiciPaolo... | lld:pubmed |
| pubmed-article:21045728 | pubmed:author | pubmed-author:DonatoLuigiL | lld:pubmed |
| pubmed-article:21045728 | pubmed:author | pubmed-author:FommeiEnzaE | lld:pubmed |
| pubmed-article:21045728 | pubmed:author | pubmed-author:LombardiMassi... | lld:pubmed |
| pubmed-article:21045728 | pubmed:author | pubmed-author:GhioneSergioS | lld:pubmed |
| pubmed-article:21045728 | pubmed:author | pubmed-author:PisaniPatrizi... | lld:pubmed |
| pubmed-article:21045728 | pubmed:author | pubmed-author:Varela-Carver... | lld:pubmed |
| pubmed-article:21045728 | pubmed:author | pubmed-author:ManciniMassim... | lld:pubmed |
| pubmed-article:21045728 | pubmed:author | pubmed-author:ParkerHowardH | lld:pubmed |
| pubmed-article:21045728 | pubmed:author | pubmed-author:D'amatiGiulia... | lld:pubmed |
| pubmed-article:21045728 | pubmed:issnType | Electronic | lld:pubmed |
| pubmed-article:21045728 | pubmed:volume | 29 | lld:pubmed |
| pubmed-article:21045728 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:21045728 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:21045728 | pubmed:pagination | 364-72 | lld:pubmed |
| pubmed-article:21045728 | pubmed:meshHeading | pubmed-meshheading:21045728... | lld:pubmed |
| pubmed-article:21045728 | pubmed:meshHeading | pubmed-meshheading:21045728... | lld:pubmed |
| pubmed-article:21045728 | pubmed:meshHeading | pubmed-meshheading:21045728... | lld:pubmed |
| pubmed-article:21045728 | pubmed:meshHeading | pubmed-meshheading:21045728... | lld:pubmed |
| pubmed-article:21045728 | pubmed:meshHeading | pubmed-meshheading:21045728... | lld:pubmed |
| pubmed-article:21045728 | pubmed:meshHeading | pubmed-meshheading:21045728... | lld:pubmed |
| pubmed-article:21045728 | pubmed:meshHeading | pubmed-meshheading:21045728... | lld:pubmed |
| pubmed-article:21045728 | pubmed:meshHeading | pubmed-meshheading:21045728... | lld:pubmed |
| pubmed-article:21045728 | pubmed:meshHeading | pubmed-meshheading:21045728... | lld:pubmed |
| pubmed-article:21045728 | pubmed:meshHeading | pubmed-meshheading:21045728... | lld:pubmed |
| pubmed-article:21045728 | pubmed:meshHeading | pubmed-meshheading:21045728... | lld:pubmed |
| pubmed-article:21045728 | pubmed:meshHeading | pubmed-meshheading:21045728... | lld:pubmed |
| pubmed-article:21045728 | pubmed:meshHeading | pubmed-meshheading:21045728... | lld:pubmed |
| pubmed-article:21045728 | pubmed:meshHeading | pubmed-meshheading:21045728... | lld:pubmed |
| pubmed-article:21045728 | pubmed:meshHeading | pubmed-meshheading:21045728... | lld:pubmed |
| pubmed-article:21045728 | pubmed:meshHeading | pubmed-meshheading:21045728... | lld:pubmed |
| pubmed-article:21045728 | pubmed:meshHeading | pubmed-meshheading:21045728... | lld:pubmed |
| pubmed-article:21045728 | pubmed:year | 2011 | lld:pubmed |
| pubmed-article:21045728 | pubmed:articleTitle | Perindopril and indapamide reverse coronary microvascular remodelling and improve flow in arterial hypertension. | lld:pubmed |
| pubmed-article:21045728 | pubmed:affiliation | Fondazione G Monasterio CNR-Regione Toscana, Pisa, Italy. | lld:pubmed |
| pubmed-article:21045728 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:21045728 | pubmed:publicationType | Clinical Trial | lld:pubmed |
| pubmed-article:21045728 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
