| pubmed-article:20942957 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:20942957 | lifeskim:mentions | umls-concept:C0178784 | lld:lifeskim |
| pubmed-article:20942957 | lifeskim:mentions | umls-concept:C0225336 | lld:lifeskim |
| pubmed-article:20942957 | lifeskim:mentions | umls-concept:C0037083 | lld:lifeskim |
| pubmed-article:20942957 | lifeskim:mentions | umls-concept:C0243026 | lld:lifeskim |
| pubmed-article:20942957 | lifeskim:mentions | umls-concept:C0277785 | lld:lifeskim |
| pubmed-article:20942957 | lifeskim:mentions | umls-concept:C0521117 | lld:lifeskim |
| pubmed-article:20942957 | lifeskim:mentions | umls-concept:C0004083 | lld:lifeskim |
| pubmed-article:20942957 | pubmed:issue | 5 | lld:pubmed |
| pubmed-article:20942957 | pubmed:dateCreated | 2010-11-24 | lld:pubmed |
| pubmed-article:20942957 | pubmed:abstractText | Previous reports suggest that endothelial activation is an important process in sepsis pathogenesis. We investigated the association between biomarkers of endothelial cell activation and sepsis severity, organ dysfunction sequential organ failure assessment (SOFA) score, and death. | lld:pubmed |
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| pubmed-article:20942957 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:20942957 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:20942957 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:20942957 | pubmed:issn | 1466-609X | lld:pubmed |
| pubmed-article:20942957 | pubmed:author | pubmed-author:NiceWW | lld:pubmed |
| pubmed-article:20942957 | pubmed:author | pubmed-author:ParikhSamir... | lld:pubmed |
| pubmed-article:20942957 | pubmed:author | pubmed-author:YanoKiichiroK | lld:pubmed |
| pubmed-article:20942957 | pubmed:author | pubmed-author:TrzeciakSteph... | lld:pubmed |
| pubmed-article:20942957 | pubmed:author | pubmed-author:AirdWilliam... | lld:pubmed |
| pubmed-article:20942957 | pubmed:author | pubmed-author:ShapiroNathan... | lld:pubmed |
| pubmed-article:20942957 | pubmed:author | pubmed-author:JonesAlan EAE | lld:pubmed |
| pubmed-article:20942957 | pubmed:author | pubmed-author:SchuetzPhilip... | lld:pubmed |
| pubmed-article:20942957 | pubmed:author | pubmed-author:SorasakiMidor... | lld:pubmed |
| pubmed-article:20942957 | pubmed:issnType | Electronic | lld:pubmed |
| pubmed-article:20942957 | pubmed:volume | 14 | lld:pubmed |
| pubmed-article:20942957 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:20942957 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:20942957 | pubmed:pagination | R182 | lld:pubmed |
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| pubmed-article:20942957 | pubmed:year | 2010 | lld:pubmed |
| pubmed-article:20942957 | pubmed:articleTitle | The association of endothelial cell signaling, severity of illness, and organ dysfunction in sepsis. | lld:pubmed |
| pubmed-article:20942957 | pubmed:affiliation | Department of Emergency Medicine, Beth Israel Deaconess Medical Center,1 Deaconess Road CC2-W, Boston, MA 02215, USA. nshapiro@bidmc.harvard.edu | lld:pubmed |
| pubmed-article:20942957 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:20942957 | pubmed:publicationType | Comparative Study | lld:pubmed |
| pubmed-article:20942957 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
| pubmed-article:20942957 | pubmed:publicationType | Research Support, N.I.H., Extramural | lld:pubmed |
| http://linkedlifedata.com/r... | pubmed:referesTo | pubmed-article:20942957 | lld:pubmed |
