Optimization of the degenerated interfacial ATP binding site improves the function of disease-related mutant cystic fibrosis transmembrane conductance regulator (CFTR) channels.

Source:http://linkedlifedata.com/resource/pubmed/id/20861014

J. Biol. Chem. 2010 Nov 26 285 48 37663-71

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PMID
20861014