pubmed-article:20811038 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:20811038 | lifeskim:mentions | umls-concept:C0086418 | lld:lifeskim |
pubmed-article:20811038 | lifeskim:mentions | umls-concept:C0021641 | lld:lifeskim |
pubmed-article:20811038 | lifeskim:mentions | umls-concept:C2610739 | lld:lifeskim |
pubmed-article:20811038 | lifeskim:mentions | umls-concept:C0851285 | lld:lifeskim |
pubmed-article:20811038 | pubmed:issue | 11 | lld:pubmed |
pubmed-article:20811038 | pubmed:dateCreated | 2010-10-28 | lld:pubmed |
pubmed-article:20811038 | pubmed:abstractText | We tested the hypothesis that an increase in insulin per se, i.e., in the absence of zinc, suppresses glucagon secretion during euglycemia and that a decrease in insulin per se stimulates glucagon secretion during hypoglycemia in humans. | lld:pubmed |
pubmed-article:20811038 | pubmed:grant | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:20811038 | pubmed:grant | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:20811038 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:20811038 | pubmed:language | eng | lld:pubmed |
pubmed-article:20811038 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:20811038 | pubmed:citationSubset | AIM | lld:pubmed |
pubmed-article:20811038 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:20811038 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:20811038 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:20811038 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:20811038 | pubmed:month | Nov | lld:pubmed |
pubmed-article:20811038 | pubmed:issn | 1939-327X | lld:pubmed |
pubmed-article:20811038 | pubmed:author | pubmed-author:CryerPhilip... | lld:pubmed |
pubmed-article:20811038 | pubmed:author | pubmed-author:CooperbergBen... | lld:pubmed |
pubmed-article:20811038 | pubmed:issnType | Electronic | lld:pubmed |
pubmed-article:20811038 | pubmed:volume | 59 | lld:pubmed |
pubmed-article:20811038 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:20811038 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:20811038 | pubmed:pagination | 2936-40 | lld:pubmed |
pubmed-article:20811038 | pubmed:dateRevised | 2011-11-17 | lld:pubmed |
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pubmed-article:20811038 | pubmed:year | 2010 | lld:pubmed |
pubmed-article:20811038 | pubmed:articleTitle | Insulin reciprocally regulates glucagon secretion in humans. | lld:pubmed |
pubmed-article:20811038 | pubmed:affiliation | Division of Endocrinology, Metabolism and Lipid Research, Washington University School of Medicine, St. Louis, Missouri, USA. | lld:pubmed |
pubmed-article:20811038 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:20811038 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
pubmed-article:20811038 | pubmed:publicationType | Research Support, N.I.H., Extramural | lld:pubmed |
http://linkedlifedata.com/r... | pubmed:referesTo | pubmed-article:20811038 | lld:pubmed |