pubmed-article:20683905 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:20683905 | lifeskim:mentions | umls-concept:C0025312 | lld:lifeskim |
pubmed-article:20683905 | lifeskim:mentions | umls-concept:C0017337 | lld:lifeskim |
pubmed-article:20683905 | lifeskim:mentions | umls-concept:C0250494 | lld:lifeskim |
pubmed-article:20683905 | lifeskim:mentions | umls-concept:C1420117 | lld:lifeskim |
pubmed-article:20683905 | lifeskim:mentions | umls-concept:C1414662 | lld:lifeskim |
pubmed-article:20683905 | lifeskim:mentions | umls-concept:C1414664 | lld:lifeskim |
pubmed-article:20683905 | lifeskim:mentions | umls-concept:C1414666 | lld:lifeskim |
pubmed-article:20683905 | lifeskim:mentions | umls-concept:C0004083 | lld:lifeskim |
pubmed-article:20683905 | pubmed:issue | 8 | lld:pubmed |
pubmed-article:20683905 | pubmed:dateCreated | 2010-8-26 | lld:pubmed |
pubmed-article:20683905 | pubmed:abstractText | Meningomyelocele (MM) results from lack of closure of the neural tube during embryologic development. Periconceptional folic acid supplementation is a modifier of MM risk in humans, leading toan interest in the folate transport genes as potential candidates for association to MM. | lld:pubmed |
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pubmed-article:20683905 | pubmed:language | eng | lld:pubmed |
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pubmed-article:20683905 | pubmed:citationSubset | IM | lld:pubmed |
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pubmed-article:20683905 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:20683905 | pubmed:month | Aug | lld:pubmed |
pubmed-article:20683905 | pubmed:issn | 1542-0760 | lld:pubmed |
pubmed-article:20683905 | pubmed:author | pubmed-author:FletcherJack... | lld:pubmed |
pubmed-article:20683905 | pubmed:author | pubmed-author:MorrisonAlann... | lld:pubmed |
pubmed-article:20683905 | pubmed:author | pubmed-author:NorthrupHopeH | lld:pubmed |
pubmed-article:20683905 | pubmed:author | pubmed-author:AuKit SingKS | lld:pubmed |
pubmed-article:20683905 | pubmed:author | pubmed-author:TyermanGayle... | lld:pubmed |
pubmed-article:20683905 | pubmed:author | pubmed-author:O'ByrneMichel... | lld:pubmed |
pubmed-article:20683905 | pubmed:author | pubmed-author:LinJone-IngJI | lld:pubmed |
pubmed-article:20683905 | pubmed:author | pubmed-author:OstermaierKat... | lld:pubmed |
pubmed-article:20683905 | pubmed:author | pubmed-author:DoebelSabineS | lld:pubmed |
pubmed-article:20683905 | pubmed:issnType | Electronic | lld:pubmed |
pubmed-article:20683905 | pubmed:volume | 88 | lld:pubmed |
pubmed-article:20683905 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:20683905 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:20683905 | pubmed:pagination | 689-94 | lld:pubmed |
pubmed-article:20683905 | pubmed:dateRevised | 2011-7-26 | lld:pubmed |
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pubmed-article:20683905 | pubmed:year | 2010 | lld:pubmed |
pubmed-article:20683905 | pubmed:articleTitle | Association of folate receptor (FOLR1, FOLR2, FOLR3) and reduced folate carrier (SLC19A1) genes with meningomyelocele. | lld:pubmed |
pubmed-article:20683905 | pubmed:affiliation | Department of Pediatrics, The University of Texas Medical School at Houston, Houston, Texas 77030, USA. | lld:pubmed |
pubmed-article:20683905 | pubmed:publicationType | Journal Article | lld:pubmed |