| pubmed-article:206644 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:206644 | lifeskim:mentions | umls-concept:C0000742 | lld:lifeskim |
| pubmed-article:206644 | lifeskim:mentions | umls-concept:C1261473 | lld:lifeskim |
| pubmed-article:206644 | lifeskim:mentions | umls-concept:C0023418 | lld:lifeskim |
| pubmed-article:206644 | lifeskim:mentions | umls-concept:C1280500 | lld:lifeskim |
| pubmed-article:206644 | pubmed:issue | 4 | lld:pubmed |
| pubmed-article:206644 | pubmed:dateCreated | 1978-7-15 | lld:pubmed |
| pubmed-article:206644 | pubmed:abstractText | Nonproducer cells transformed by Kirsten sarcoma virus (KiSV) or Abelson murine leukemia virus (A-MuLV) were infected with N- or NB-tropic helper viruses to rescue the defective transforming virus. The titer of the transforming viruses was determined on NIH/3T3 fibroblast-like cells and cell-free filtrates of virus stock were inoculated into newborn Fv-1nn mice. Friend, Moloney, and Rauscher group of MuLV (FMR) pseudotypes of KiSV induced an erythroid leukemia efficiently, while an endogenous helper (N35-MuLV) pseudotype of KiSV did not. FMR pseudotypes of A-MuLV induced the Abelson lymphoid leukemia, while the N35-MuLV or a Kirsten leukemia virus (Ki-MuLV) pseudotype did not. Pseudotypes of A-MuLV were used to infect bone marrow cells of Fv-1nn mice in vitro. The FMR pseudotypes transformed bone marrow cells at 40-100-fold higher frequency than the N35-MuLV or Ki-MuLV pseudotypes. Mixing experiments demonstrated that the addition of an effective helper, such as M-MuLV did not enhance lymphoid transformation by ineffective A-MuLV (N35-MuLV). The A-MuLV genome is responsible for hematopoietic cell transformation because a nonproducer clone of lymphoid cells, free of helper virus, was isolated. The data indicates that the pseudotype of A-MuLV determines its ability to transform hematopoietic cells. | lld:pubmed |
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| pubmed-article:206644 | pubmed:language | eng | lld:pubmed |
| pubmed-article:206644 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:206644 | pubmed:citationSubset | AIM | lld:pubmed |
| pubmed-article:206644 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:206644 | pubmed:month | Apr | lld:pubmed |
| pubmed-article:206644 | pubmed:issn | 0022-1007 | lld:pubmed |
| pubmed-article:206644 | pubmed:author | pubmed-author:ScherC DCD | lld:pubmed |
| pubmed-article:206644 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:206644 | pubmed:day | 1 | lld:pubmed |
| pubmed-article:206644 | pubmed:volume | 147 | lld:pubmed |
| pubmed-article:206644 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:206644 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:206644 | pubmed:pagination | 1044-53 | lld:pubmed |
| pubmed-article:206644 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
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| pubmed-article:206644 | pubmed:year | 1978 | lld:pubmed |
| pubmed-article:206644 | pubmed:articleTitle | Effect of pseudotype on Abelson virus and Kirsten sarcoma virus-induced leukemia. | lld:pubmed |
| pubmed-article:206644 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:206644 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
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