pubmed-article:20662716 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:20662716 | lifeskim:mentions | umls-concept:C0019704 | lld:lifeskim |
pubmed-article:20662716 | lifeskim:mentions | umls-concept:C0020971 | lld:lifeskim |
pubmed-article:20662716 | lifeskim:mentions | umls-concept:C0027361 | lld:lifeskim |
pubmed-article:20662716 | lifeskim:mentions | umls-concept:C0008976 | lld:lifeskim |
pubmed-article:20662716 | lifeskim:mentions | umls-concept:C0033684 | lld:lifeskim |
pubmed-article:20662716 | lifeskim:mentions | umls-concept:C0042210 | lld:lifeskim |
pubmed-article:20662716 | lifeskim:mentions | umls-concept:C0444669 | lld:lifeskim |
pubmed-article:20662716 | lifeskim:mentions | umls-concept:C0884118 | lld:lifeskim |
pubmed-article:20662716 | lifeskim:mentions | umls-concept:C1318970 | lld:lifeskim |
pubmed-article:20662716 | pubmed:issue | 5 | lld:pubmed |
pubmed-article:20662716 | pubmed:dateCreated | 2010-8-5 | lld:pubmed |
pubmed-article:20662716 | pubmed:abstractText | Human immunodeficiency virus type 1 (HIV-1)-specific cellular immunity contributes to the control of HIV-1 replication. HIV-1-infected volunteers who were receiving antiretroviral therapy were given a replication-defective adenovirus type 5 HIV-1 gag vaccine in a randomized, blinded therapeutic vaccination study. | lld:pubmed |
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pubmed-article:20662716 | pubmed:language | eng | lld:pubmed |
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