| pubmed-article:20604685 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:20604685 | lifeskim:mentions | umls-concept:C0314657 | lld:lifeskim |
| pubmed-article:20604685 | lifeskim:mentions | umls-concept:C0178468 | lld:lifeskim |
| pubmed-article:20604685 | lifeskim:mentions | umls-concept:C0016884 | lld:lifeskim |
| pubmed-article:20604685 | pubmed:issue | 7 | lld:pubmed |
| pubmed-article:20604685 | pubmed:dateCreated | 2010-7-7 | lld:pubmed |
| pubmed-article:20604685 | pubmed:abstractText | Autoimmune thyroid diseases (AITD), including Graves' disease and Hashimoto's thyroiditis, arise due to complex interactions between environmental and genetic factors. There are sound data coming from epidemiological, family, and twin studies demonstrating a strong genetic influence on the development of AITD. In this review we summarize the new findings on the genetic susceptibility to AITD focusing on emerging mechanisms of susceptibility. | lld:pubmed |
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