| pubmed-article:20595778 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:20595778 | lifeskim:mentions | umls-concept:C1135183 | lld:lifeskim |
| pubmed-article:20595778 | lifeskim:mentions | umls-concept:C0026336 | lld:lifeskim |
| pubmed-article:20595778 | lifeskim:mentions | umls-concept:C0014257 | lld:lifeskim |
| pubmed-article:20595778 | lifeskim:mentions | umls-concept:C0542341 | lld:lifeskim |
| pubmed-article:20595778 | lifeskim:mentions | umls-concept:C1280500 | lld:lifeskim |
| pubmed-article:20595778 | lifeskim:mentions | umls-concept:C1155266 | lld:lifeskim |
| pubmed-article:20595778 | lifeskim:mentions | umls-concept:C1322815 | lld:lifeskim |
| pubmed-article:20595778 | pubmed:issue | 8 | lld:pubmed |
| pubmed-article:20595778 | pubmed:dateCreated | 2010-7-30 | lld:pubmed |
| pubmed-article:20595778 | pubmed:abstractText | The rate of stent thrombosis is increased in association with drug-eluting stents (DES) due to delayed endothelialization and prolonged inflammation. Clinical studies have shown that either an angiotensin-receptor blocker (ARB) or a calcium-channel blocker (CCB) can improve endothelial dysfunction and inhibit inflammatory reactions in patients with hypertension. The effects of co-administered CCB and ARB on vascular protection after DES implantation, however, remain unknown. | lld:pubmed |
| pubmed-article:20595778 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:20595778 | pubmed:language | eng | lld:pubmed |
| pubmed-article:20595778 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:20595778 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:20595778 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:20595778 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:20595778 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:20595778 | pubmed:month | Aug | lld:pubmed |
| pubmed-article:20595778 | pubmed:issn | 1347-4820 | lld:pubmed |
| pubmed-article:20595778 | pubmed:author | pubmed-author:MiyauchiKatsu... | lld:pubmed |
| pubmed-article:20595778 | pubmed:author | pubmed-author:DaidaHiroyuki... | lld:pubmed |
| pubmed-article:20595778 | pubmed:author | pubmed-author:IesakiTakafum... | lld:pubmed |
| pubmed-article:20595778 | pubmed:author | pubmed-author:SumiyoshiKats... | lld:pubmed |
| pubmed-article:20595778 | pubmed:author | pubmed-author:KajimotoKanK | lld:pubmed |
| pubmed-article:20595778 | pubmed:author | pubmed-author:KasaiTakatosh... | lld:pubmed |
| pubmed-article:20595778 | pubmed:author | pubmed-author:DohiTomotakaT | lld:pubmed |
| pubmed-article:20595778 | pubmed:author | pubmed-author:KubotaNaozumi... | lld:pubmed |
| pubmed-article:20595778 | pubmed:issnType | Electronic | lld:pubmed |
| pubmed-article:20595778 | pubmed:volume | 74 | lld:pubmed |
| pubmed-article:20595778 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:20595778 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:20595778 | pubmed:pagination | 1704-10 | lld:pubmed |
| pubmed-article:20595778 | pubmed:meshHeading | pubmed-meshheading:20595778... | lld:pubmed |
| pubmed-article:20595778 | pubmed:meshHeading | pubmed-meshheading:20595778... | lld:pubmed |
| pubmed-article:20595778 | pubmed:meshHeading | pubmed-meshheading:20595778... | lld:pubmed |
| pubmed-article:20595778 | pubmed:meshHeading | pubmed-meshheading:20595778... | lld:pubmed |
| pubmed-article:20595778 | pubmed:meshHeading | pubmed-meshheading:20595778... | lld:pubmed |
| pubmed-article:20595778 | pubmed:meshHeading | pubmed-meshheading:20595778... | lld:pubmed |
| pubmed-article:20595778 | pubmed:meshHeading | pubmed-meshheading:20595778... | lld:pubmed |
| pubmed-article:20595778 | pubmed:meshHeading | pubmed-meshheading:20595778... | lld:pubmed |
| pubmed-article:20595778 | pubmed:meshHeading | pubmed-meshheading:20595778... | lld:pubmed |
| pubmed-article:20595778 | pubmed:year | 2010 | lld:pubmed |
| pubmed-article:20595778 | pubmed:articleTitle | Synergistic effects of calcium-channel and angiotensin-receptor blockers on endothelial function and inflammatory responses in a porcine drug-eluting stent model. | lld:pubmed |
| pubmed-article:20595778 | pubmed:affiliation | Department of Cardiology, Juntendo University School of Medicine, Tokyo, Japan. | lld:pubmed |
| pubmed-article:20595778 | pubmed:publicationType | Journal Article | lld:pubmed |
| http://linkedlifedata.com/r... | pubmed:referesTo | pubmed-article:20595778 | lld:pubmed |
