| pubmed-article:20533870 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:20533870 | lifeskim:mentions | umls-concept:C0079488 | lld:lifeskim |
| pubmed-article:20533870 | lifeskim:mentions | umls-concept:C0806987 | lld:lifeskim |
| pubmed-article:20533870 | lifeskim:mentions | umls-concept:C0205177 | lld:lifeskim |
| pubmed-article:20533870 | lifeskim:mentions | umls-concept:C1327414 | lld:lifeskim |
| pubmed-article:20533870 | lifeskim:mentions | umls-concept:C0376315 | lld:lifeskim |
| pubmed-article:20533870 | lifeskim:mentions | umls-concept:C1882417 | lld:lifeskim |
| pubmed-article:20533870 | lifeskim:mentions | umls-concept:C0205263 | lld:lifeskim |
| pubmed-article:20533870 | pubmed:issue | 2 | lld:pubmed |
| pubmed-article:20533870 | pubmed:dateCreated | 2010-6-21 | lld:pubmed |
| pubmed-article:20533870 | pubmed:abstractText | Infection with Helicobacter pylori possessing a newly described virulence factor--duodenal ulcer-promoting gene A (dupA)--has been associated with duodenal ulceration and increased gastric inflammation. | lld:pubmed |
| pubmed-article:20533870 | pubmed:grant | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:20533870 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:20533870 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:20533870 | pubmed:citationSubset | AIM | lld:pubmed |
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| pubmed-article:20533870 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:20533870 | pubmed:month | Jul | lld:pubmed |
| pubmed-article:20533870 | pubmed:issn | 1537-6613 | lld:pubmed |
| pubmed-article:20533870 | pubmed:author | pubmed-author:AthertonJohn... | lld:pubmed |
| pubmed-article:20533870 | pubmed:author | pubmed-author:RobinsonKaren... | lld:pubmed |
| pubmed-article:20533870 | pubmed:author | pubmed-author:ArgentRichard... | lld:pubmed |
| pubmed-article:20533870 | pubmed:author | pubmed-author:HusseinNawfal... | lld:pubmed |
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| pubmed-article:20533870 | pubmed:author | pubmed-author:PatelSapna... | lld:pubmed |
| pubmed-article:20533870 | pubmed:issnType | Electronic | lld:pubmed |
| pubmed-article:20533870 | pubmed:day | 15 | lld:pubmed |
| pubmed-article:20533870 | pubmed:volume | 202 | lld:pubmed |
| pubmed-article:20533870 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:20533870 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:20533870 | pubmed:pagination | 261-9 | lld:pubmed |
| pubmed-article:20533870 | pubmed:dateRevised | 2011-4-26 | lld:pubmed |
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| pubmed-article:20533870 | pubmed:year | 2010 | lld:pubmed |
| pubmed-article:20533870 | pubmed:articleTitle | Helicobacter pylori dupA is polymorphic, and its active form induces proinflammatory cytokine secretion by mononuclear cells. | lld:pubmed |
| pubmed-article:20533870 | pubmed:affiliation | Nottingham Digestive Diseases Centre Biomedical Research Unit, Queen's Medical Centre, University of Nottingham, Nottingham, United Kingdom. | lld:pubmed |
| pubmed-article:20533870 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:20533870 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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| http://linkedlifedata.com/r... | pubmed:referesTo | pubmed-article:20533870 | lld:pubmed |
