Linked Life Data

20533393

Source:http://linkedlifedata.com/resource/pubmed/id/20533393

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pubmed-article:20533393pubmed:abstractTextIntravascular hemolysis may cause tissue injury directly or via a systemic in?ammatory response. Under physiological conditions, extracorpuscular hemoglobin (Hb) is bound by haptoglobin(Hp) and the complex internalized via the hemoglobin scavenger receptor CD163 on monocytes, prior to catabolism via heme-oxygenase-1 (HO-1). Recently, a novel subset of CD68(pos)CD163(high)HLA-DR(low) macrophages with high expression of HO-1 was recognized in hemorrhagic areas of atherosclerotic plaques, distinct from CD68(pos)CD163(low)HLA-DR(high) foam cell macrophages with low- HO-1 content. Considering the hemolytic insult during CPB, we hypothesized that an equivalent compensatory CD163(high)HLA-DR(low) phenotype will evolve in circulating CD14(pos) monocytes post surgery.lld:pubmed
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pubmed-article:20533393pubmed:copyrightInfo© 2010 International Clinical Cytometry Societylld:pubmed
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pubmed-article:20533393pubmed:volume78lld:pubmed
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pubmed-article:20533393pubmed:year2010lld:pubmed
pubmed-article:20533393pubmed:articleTitlePhenotypic commitment of monocytes towards a protective hemoglobin scavenging phenotype (CD14(pos)CD163(high)HLA-DR(low))following cardiopulmonary bypass.lld:pubmed
pubmed-article:20533393pubmed:affiliationEdmund Cohen Laboratory for Vascular Research, Chronic Disease Research Centre, Tropical Medicine Research Institute, University of the West Indies, Barbados.lld:pubmed
pubmed-article:20533393pubmed:publicationTypeJournal Articlelld:pubmed