| pubmed-article:2047861 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:2047861 | lifeskim:mentions | umls-concept:C0242299 | lld:lifeskim |
| pubmed-article:2047861 | lifeskim:mentions | umls-concept:C0013030 | lld:lifeskim |
| pubmed-article:2047861 | lifeskim:mentions | umls-concept:C0034840 | lld:lifeskim |
| pubmed-article:2047861 | lifeskim:mentions | umls-concept:C1879547 | lld:lifeskim |
| pubmed-article:2047861 | lifeskim:mentions | umls-concept:C1883220 | lld:lifeskim |
| pubmed-article:2047861 | pubmed:issue | 5012 | lld:pubmed |
| pubmed-article:2047861 | pubmed:dateCreated | 1991-7-12 | lld:pubmed |
| pubmed-article:2047861 | pubmed:abstractText | The chicken ovalbumin upstream promoter transcription factor (COUP-TF) is a member of the steroid receptor superfamily and participates in the regulation of several genes. While a number of functions have been ascribed to COUP-TF, no ligand or activator molecule has been identified, and thus it is classified as one of a group of orphan receptors. Activation of COUP-TF by physiological concentrations of the neurotransmitter dopamine was observed in transient transfection assays. Treatment of transfected cells with the dopamine receptor agonist alpha-ergocryptine also activated COUP-dependent expression of a reporter gene. COUP-TF that contained a deletion in the COOH-terminal domain was not activated by these compounds. These observations suggest that dopamine may be a physiological activator of COUP-TF. | lld:pubmed |
| pubmed-article:2047861 | pubmed:language | eng | lld:pubmed |
| pubmed-article:2047861 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:2047861 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:2047861 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:2047861 | pubmed:month | Jun | lld:pubmed |
| pubmed-article:2047861 | pubmed:issn | 0036-8075 | lld:pubmed |
| pubmed-article:2047861 | pubmed:author | pubmed-author:O'MalleyB WBW | lld:pubmed |
| pubmed-article:2047861 | pubmed:author | pubmed-author:ConneelyO MOM | lld:pubmed |
| pubmed-article:2047861 | pubmed:author | pubmed-author:PowerR FRF | lld:pubmed |
| pubmed-article:2047861 | pubmed:author | pubmed-author:LydonJ PJP | lld:pubmed |
| pubmed-article:2047861 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:2047861 | pubmed:day | 14 | lld:pubmed |
| pubmed-article:2047861 | pubmed:volume | 252 | lld:pubmed |
| pubmed-article:2047861 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:2047861 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:2047861 | pubmed:pagination | 1546-8 | lld:pubmed |
| pubmed-article:2047861 | pubmed:dateRevised | 2008-11-21 | lld:pubmed |
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| pubmed-article:2047861 | pubmed:year | 1991 | lld:pubmed |
| pubmed-article:2047861 | pubmed:articleTitle | Dopamine activation of an orphan of the steroid receptor superfamily. | lld:pubmed |
| pubmed-article:2047861 | pubmed:affiliation | Department of Cell Biology, Baylor College of Medicine, Houston, TX 77030. | lld:pubmed |
| pubmed-article:2047861 | pubmed:publicationType | Journal Article | lld:pubmed |
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