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| pubmed-article:20421849 | lifeskim:mentions | umls-concept:C0221711 | lld:lifeskim |
| pubmed-article:20421849 | lifeskim:mentions | umls-concept:C1099474 | lld:lifeskim |
| pubmed-article:20421849 | lifeskim:mentions | umls-concept:C1415300 | lld:lifeskim |
| pubmed-article:20421849 | lifeskim:mentions | umls-concept:C0871125 | lld:lifeskim |
| pubmed-article:20421849 | lifeskim:mentions | umls-concept:C1882417 | lld:lifeskim |
| pubmed-article:20421849 | lifeskim:mentions | umls-concept:C0392760 | lld:lifeskim |
| pubmed-article:20421849 | lifeskim:mentions | umls-concept:C1711351 | lld:lifeskim |
| pubmed-article:20421849 | pubmed:issue | 5 | lld:pubmed |
| pubmed-article:20421849 | pubmed:dateCreated | 2010-9-6 | lld:pubmed |
| pubmed-article:20421849 | pubmed:abstractText | To identify the reliable connectivity between causal genes or variants with an abnormality expressed in a certain endophenotype has been viewed as a crucial step in unraveling the etiology of schizophrenia because of the considerable heterogeneity in this disorder. | lld:pubmed |
| pubmed-article:20421849 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:20421849 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:20421849 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:20421849 | pubmed:month | Oct | lld:pubmed |
| pubmed-article:20421849 | pubmed:issn | 1473-5873 | lld:pubmed |
| pubmed-article:20421849 | pubmed:author | pubmed-author:KeyM KMK | lld:pubmed |
| pubmed-article:20421849 | pubmed:author | pubmed-author:GlattStephen... | lld:pubmed |
| pubmed-article:20421849 | pubmed:author | pubmed-author:YonedaHiroshi... | lld:pubmed |
| pubmed-article:20421849 | pubmed:author | pubmed-author:MotomuraNaoya... | lld:pubmed |
| pubmed-article:20421849 | pubmed:author | pubmed-author:TsutsumiAtsus... | lld:pubmed |
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| pubmed-article:20421849 | pubmed:author | pubmed-author:MatsumuraHito... | lld:pubmed |
| pubmed-article:20421849 | pubmed:author | pubmed-author:KanazawaTetsu... | lld:pubmed |
| pubmed-article:20421849 | pubmed:author | pubmed-author:UenishiHiroyu... | lld:pubmed |
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| pubmed-article:20421849 | pubmed:author | pubmed-author:HokyoAkiraA | lld:pubmed |
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| pubmed-article:20421849 | pubmed:issnType | Electronic | lld:pubmed |
| pubmed-article:20421849 | pubmed:volume | 20 | lld:pubmed |
| pubmed-article:20421849 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:20421849 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:20421849 | pubmed:pagination | 191-8 | lld:pubmed |
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| pubmed-article:20421849 | pubmed:year | 2010 | lld:pubmed |
| pubmed-article:20421849 | pubmed:articleTitle | Habituation in prepulse inhibition is affected by a polymorphism on the NMDA receptor 2B subunit gene (GRIN2B). | lld:pubmed |
| pubmed-article:20421849 | pubmed:affiliation | Department of Neuropsychiatry, Osaka Medical College, Takatsuki, Osaka, Japan. | lld:pubmed |
| pubmed-article:20421849 | pubmed:publicationType | Journal Article | lld:pubmed |
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