| pubmed-article:20417490 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:20417490 | lifeskim:mentions | umls-concept:C0000768 | lld:lifeskim |
| pubmed-article:20417490 | lifeskim:mentions | umls-concept:C0013935 | lld:lifeskim |
| pubmed-article:20417490 | lifeskim:mentions | umls-concept:C1333573 | lld:lifeskim |
| pubmed-article:20417490 | lifeskim:mentions | umls-concept:C1333633 | lld:lifeskim |
| pubmed-article:20417490 | lifeskim:mentions | umls-concept:C0021467 | lld:lifeskim |
| pubmed-article:20417490 | lifeskim:mentions | umls-concept:C0021469 | lld:lifeskim |
| pubmed-article:20417490 | lifeskim:mentions | umls-concept:C0059438 | lld:lifeskim |
| pubmed-article:20417490 | lifeskim:mentions | umls-concept:C1879547 | lld:lifeskim |
| pubmed-article:20417490 | pubmed:issue | 1 | lld:pubmed |
| pubmed-article:20417490 | pubmed:dateCreated | 2010-7-8 | lld:pubmed |
| pubmed-article:20417490 | pubmed:abstractText | Maternal hyperglycemia increases the risk of congenital malformations. Epigallocatechin-3-gallate (EGCG), a natural antioxidant purified from green tea, inhibits oxidative stress signaling. We propose that EGCG prevents hyperglycemia-induced malformation via inhibition of oxidative stress signaling. The objective of this study is to examine the effect of EGCG on hyperglycemia-induced adverse effects during embryonic development. | lld:pubmed |
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| pubmed-article:20417490 | pubmed:language | eng | lld:pubmed |
| pubmed-article:20417490 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:20417490 | pubmed:citationSubset | AIM | lld:pubmed |
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| pubmed-article:20417490 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:20417490 | pubmed:month | Jul | lld:pubmed |
| pubmed-article:20417490 | pubmed:issn | 1097-6868 | lld:pubmed |
| pubmed-article:20417490 | pubmed:author | pubmed-author:FryDD | lld:pubmed |
| pubmed-article:20417490 | pubmed:author | pubmed-author:YangPeixinP | lld:pubmed |
| pubmed-article:20417490 | pubmed:copyrightInfo | Copyright (c) 2010 Mosby, Inc. All rights reserved. | lld:pubmed |
| pubmed-article:20417490 | pubmed:issnType | Electronic | lld:pubmed |
| pubmed-article:20417490 | pubmed:volume | 203 | lld:pubmed |
| pubmed-article:20417490 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:20417490 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:20417490 | pubmed:pagination | 75.e1-6 | lld:pubmed |
| pubmed-article:20417490 | pubmed:dateRevised | 2011-8-1 | lld:pubmed |
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| pubmed-article:20417490 | pubmed:year | 2010 | lld:pubmed |
| pubmed-article:20417490 | pubmed:articleTitle | Epigallocatechin-3-gallate ameliorates hyperglycemia-induced embryonic vasculopathy and malformation by inhibition of Foxo3a activation. | lld:pubmed |
| pubmed-article:20417490 | pubmed:affiliation | Department of Obstetrics, Gynecology, and Reproductive Sciences, University of Maryland School of Medicine, Baltimore, MD, USA. | lld:pubmed |
| pubmed-article:20417490 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:20417490 | pubmed:publicationType | In Vitro | lld:pubmed |
| pubmed-article:20417490 | pubmed:publicationType | Research Support, N.I.H., Extramural | lld:pubmed |
