| pubmed-article:20363318 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:20363318 | lifeskim:mentions | umls-concept:C0934502 | lld:lifeskim |
| pubmed-article:20363318 | lifeskim:mentions | umls-concept:C0079904 | lld:lifeskim |
| pubmed-article:20363318 | lifeskim:mentions | umls-concept:C0033666 | lld:lifeskim |
| pubmed-article:20363318 | lifeskim:mentions | umls-concept:C0086982 | lld:lifeskim |
| pubmed-article:20363318 | lifeskim:mentions | umls-concept:C0851285 | lld:lifeskim |
| pubmed-article:20363318 | pubmed:issue | 9 | lld:pubmed |
| pubmed-article:20363318 | pubmed:dateCreated | 2010-6-28 | lld:pubmed |
| pubmed-article:20363318 | pubmed:abstractText | The eukaryotic transcription factor NF-kappaB regulates a wide range of host genes that control the inflammatory and immune responses, programmed cell death, cell proliferation and differentiation. The activation of NF-kappaB is tightly controlled both in the cytoplasm and in the nucleus. While the upstream cytoplasmic regulatory events for the activation of NF-kappaB are well studied, much less is known about the nuclear regulation of NF-kappaB. Emerging evidence suggests that NF-kappaB undergoes a variety of posttranslational modifications, and that these modifications play a key role in determining the duration and strength of NF-kappaB nuclear activity as well as its transcriptional output. Here we summarize the recent advances in our understanding of the posttranslational modifications of NF-kappaB, the interplay between the various modifications, and the physiological relevance of these modifications. | lld:pubmed |
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