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pubmed-article:20362415pubmed:dateCreated2010-5-25lld:pubmed
pubmed-article:20362415pubmed:abstractTextThe mechanism of sudden death in hypertrophic cardiomyopathy (HCM) is ventricular tachyarrhythmia emanating from myocyte disarray, fibrosis, and inhomogeneity in intramyocardial activation. Tissue synchronization imaging (TSI) allows the measurement of regional delay, while two-dimensional strain can be used to identify myocardial fibrosis. The aim of this study was to assess the relationship between new ultrasonically derived parameters and nonsustained ventricular tachycardia (NSVT) in patients with HCM.lld:pubmed
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pubmed-article:20362415pubmed:copyrightInfoCopyright 2010 American Society of Echocardiography. Published by Mosby, Inc. All rights reserved.lld:pubmed
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pubmed-article:20362415pubmed:articleTitleNon sustained ventricular tachycardia in hypertrophic cardiomyopathy and new ultrasonic derived parameters.lld:pubmed
pubmed-article:20362415pubmed:affiliationSecond University of Naples, Chair of Cardiology, Naples, Italy. giodisal@yahoo.itlld:pubmed
pubmed-article:20362415pubmed:publicationTypeJournal Articlelld:pubmed
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