| pubmed-article:20228930 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:20228930 | lifeskim:mentions | umls-concept:C0035820 | lld:lifeskim |
| pubmed-article:20228930 | lifeskim:mentions | umls-concept:C0004096 | lld:lifeskim |
| pubmed-article:20228930 | lifeskim:mentions | umls-concept:C0021368 | lld:lifeskim |
| pubmed-article:20228930 | lifeskim:mentions | umls-concept:C0458827 | lld:lifeskim |
| pubmed-article:20228930 | lifeskim:mentions | umls-concept:C2936411 | lld:lifeskim |
| pubmed-article:20228930 | lifeskim:mentions | umls-concept:C0963088 | lld:lifeskim |
| pubmed-article:20228930 | pubmed:issue | 1 | lld:pubmed |
| pubmed-article:20228930 | pubmed:dateCreated | 2010-3-15 | lld:pubmed |
| pubmed-article:20228930 | pubmed:abstractText | Asthma is characterized by chronic airway inflammation with intense eosinophil and lymphocyte infiltration, mucus hyperproduction, and airway hyperresponsiveness. Accumulating evidence indicates that antigen-specific Th2 cells and their cytokines such as IL-4, IL-5, and IL-13 orchestrate these pathognomonic features of asthma. In addition, we and others have recently shown that IL-17-producing CD4(+) T cells (Th17 cells) and IL-23, an IL-12-related cytokine that is essential for survival and functional maturation of Th17 cells, are involved in antigen-induced airway inflammation. In this review, our current understanding of the roles of IL-23 and Th17 cells in the pathogenesis of allergic airway inflammation will be summarized. | lld:pubmed |
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| pubmed-article:20228930 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
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| pubmed-article:20228930 | pubmed:language | eng | lld:pubmed |
| pubmed-article:20228930 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:20228930 | pubmed:status | PubMed-not-MEDLINE | lld:pubmed |
| pubmed-article:20228930 | pubmed:month | Feb | lld:pubmed |
| pubmed-article:20228930 | pubmed:issn | 2092-6685 | lld:pubmed |
| pubmed-article:20228930 | pubmed:author | pubmed-author:NakajimaHiros... | lld:pubmed |
| pubmed-article:20228930 | pubmed:author | pubmed-author:HiroseKoichiK | lld:pubmed |
| pubmed-article:20228930 | pubmed:issnType | Electronic | lld:pubmed |
| pubmed-article:20228930 | pubmed:volume | 10 | lld:pubmed |
| pubmed-article:20228930 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:20228930 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:20228930 | pubmed:pagination | 1-4 | lld:pubmed |
| pubmed-article:20228930 | pubmed:year | 2010 | lld:pubmed |
| pubmed-article:20228930 | pubmed:articleTitle | Role of IL-23 and Th17 Cells in Airway Inflammation in Asthma. | lld:pubmed |
| pubmed-article:20228930 | pubmed:affiliation | Department of Molecular Genetics, Graduate School of Medicine, Chiba University, Chiba, Japan. | lld:pubmed |
| pubmed-article:20228930 | pubmed:publicationType | Journal Article | lld:pubmed |
| http://linkedlifedata.com/r... | pubmed:referesTo | pubmed-article:20228930 | lld:pubmed |
