pubmed-article:202260 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:202260 | lifeskim:mentions | umls-concept:C0014442 | lld:lifeskim |
pubmed-article:202260 | lifeskim:mentions | umls-concept:C1882726 | lld:lifeskim |
pubmed-article:202260 | lifeskim:mentions | umls-concept:C0038432 | lld:lifeskim |
pubmed-article:202260 | lifeskim:mentions | umls-concept:C0441655 | lld:lifeskim |
pubmed-article:202260 | lifeskim:mentions | umls-concept:C0011847 | lld:lifeskim |
pubmed-article:202260 | lifeskim:mentions | umls-concept:C1280500 | lld:lifeskim |
pubmed-article:202260 | lifeskim:mentions | umls-concept:C1157332 | lld:lifeskim |
pubmed-article:202260 | lifeskim:mentions | umls-concept:C0205263 | lld:lifeskim |
pubmed-article:202260 | lifeskim:mentions | umls-concept:C0205191 | lld:lifeskim |
pubmed-article:202260 | pubmed:issue | 2 | lld:pubmed |
pubmed-article:202260 | pubmed:dateCreated | 1978-2-23 | lld:pubmed |
pubmed-article:202260 | pubmed:abstractText | 1. Rats were injected with a single dose of 35mg of streptozotocin/kg body wt. They exhibited a diabetes that was characterized by glycosuria, polyuria, polydipsia, hyperphagia, hyperglycaemia, increased concentrations of unesterified fatty acids, glycerol and triacylglycerols in the serum and an increased activity of glucose 6-phosphatase in the liver. 2. After 10 weeks the hepatic activities of the microsomal glycerol phosphate acyltransferase, phosphatidate phosphohydrolase, phosphatidate cytidylyltransferase, diacylglycerol acyltransferase, choline phosphotransferase, CDP-diacylglycerol--inositol phosphatidyltransferase and the soluble phosphatidate phosphohydrolase were measured. 3. The only significant changes were an increase in the activity of the soluble phosphatidate phosphohydrolase and a decrease in that of the CDP-diacylglycerol--inositol phosphatidyltransferase in the diabetic rats. 4. These results are discussed in relation to the control of glycerolipid synthesis. | lld:pubmed |
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pubmed-article:202260 | pubmed:language | eng | lld:pubmed |
pubmed-article:202260 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:202260 | pubmed:citationSubset | IM | lld:pubmed |
pubmed-article:202260 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
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pubmed-article:202260 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
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pubmed-article:202260 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:202260 | pubmed:month | Nov | lld:pubmed |
pubmed-article:202260 | pubmed:issn | 0264-6021 | lld:pubmed |
pubmed-article:202260 | pubmed:author | pubmed-author:BrindleyD NDN | lld:pubmed |
pubmed-article:202260 | pubmed:author | pubmed-author:BowleyMM | lld:pubmed |
pubmed-article:202260 | pubmed:author | pubmed-author:HawthorneJ... | lld:pubmed |
pubmed-article:202260 | pubmed:author | pubmed-author:PritchardP... | lld:pubmed |
pubmed-article:202260 | pubmed:author | pubmed-author:WhitingP HPH | lld:pubmed |
pubmed-article:202260 | pubmed:author | pubmed-author:SturtonR GRG | lld:pubmed |
pubmed-article:202260 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:202260 | pubmed:day | 15 | lld:pubmed |
pubmed-article:202260 | pubmed:volume | 168 | lld:pubmed |
pubmed-article:202260 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:202260 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:202260 | pubmed:pagination | 147-53 | lld:pubmed |
pubmed-article:202260 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
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