pubmed-article:20164142 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:20164142 | lifeskim:mentions | umls-concept:C0242724 | lld:lifeskim |
pubmed-article:20164142 | lifeskim:mentions | umls-concept:C0023980 | lld:lifeskim |
pubmed-article:20164142 | lifeskim:mentions | umls-concept:C0035820 | lld:lifeskim |
pubmed-article:20164142 | lifeskim:mentions | umls-concept:C0032214 | lld:lifeskim |
pubmed-article:20164142 | lifeskim:mentions | umls-concept:C0600138 | lld:lifeskim |
pubmed-article:20164142 | lifeskim:mentions | umls-concept:C1704632 | lld:lifeskim |
pubmed-article:20164142 | lifeskim:mentions | umls-concept:C0871261 | lld:lifeskim |
pubmed-article:20164142 | lifeskim:mentions | umls-concept:C2911692 | lld:lifeskim |
pubmed-article:20164142 | lifeskim:mentions | umls-concept:C1706817 | lld:lifeskim |
pubmed-article:20164142 | lifeskim:mentions | umls-concept:C1521761 | lld:lifeskim |
pubmed-article:20164142 | lifeskim:mentions | umls-concept:C0004409 | lld:lifeskim |
pubmed-article:20164142 | lifeskim:mentions | umls-concept:C0205164 | lld:lifeskim |
pubmed-article:20164142 | lifeskim:mentions | umls-concept:C1998811 | lld:lifeskim |
pubmed-article:20164142 | pubmed:issue | 5 | lld:pubmed |
pubmed-article:20164142 | pubmed:dateCreated | 2010-3-15 | lld:pubmed |
pubmed-article:20164142 | pubmed:abstractText | Auxin regulates a variety of physiological and developmental processes in plants. Although auxin acts as a suppressor of leaf senescence, its exact role in this respect has not been clearly defined, aside from circumstantial evidence. It was found here that ARF2 functions in the auxin-mediated control of Arabidopsis leaf longevity, as discovered by screening EMS mutant pools for a delayed leaf senescence phenotype. Two allelic mutations, ore14-1 and 14-2, caused a highly significant delay in all senescence parameters examined, including chlorophyll content, the photochemical efficiency of photosystem II, membrane ion leakage, and the expression of senescence-associated genes. A delay of senescence symptoms was also observed under various senescence-accelerating conditions, where detached leaves were treated with darkness, phytohormones, or oxidative stress. These results indicate that the gene defined by these mutations might be a key regulatory genetic component controlling functional leaf senescence. Map-based cloning of ORE14 revealed that it encodes ARF2, a member of the auxin response factor (ARF) protein family, which modulates early auxin-induced gene expression in plants. The ore14/arf2 mutation also conferred an increased sensitivity to exogenous auxin in hypocotyl growth inhibition, thereby demonstrating that ARF2 is a repressor of auxin signalling. Therefore, the ore14/arf2 lesion appears to cause reduced repression of auxin signalling with increased auxin sensitivity, leading to delayed senescence. Altogether, our data suggest that ARF2 positively regulates leaf senescence in Arabidopsis. | lld:pubmed |
pubmed-article:20164142 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:20164142 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:20164142 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:20164142 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:20164142 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:20164142 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:20164142 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:20164142 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:20164142 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:20164142 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:20164142 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:20164142 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:20164142 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:20164142 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:20164142 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:20164142 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:20164142 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:20164142 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:20164142 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:20164142 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:20164142 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:20164142 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:20164142 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:20164142 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:20164142 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:20164142 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:20164142 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:20164142 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:20164142 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:20164142 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:20164142 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:20164142 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:20164142 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:20164142 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:20164142 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:20164142 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:20164142 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:20164142 | pubmed:language | eng | lld:pubmed |
pubmed-article:20164142 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:20164142 | pubmed:citationSubset | IM | lld:pubmed |
pubmed-article:20164142 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:20164142 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:20164142 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:20164142 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:20164142 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:20164142 | pubmed:month | Mar | lld:pubmed |
pubmed-article:20164142 | pubmed:issn | 1460-2431 | lld:pubmed |
pubmed-article:20164142 | pubmed:author | pubmed-author:NamHong GilHG | lld:pubmed |
pubmed-article:20164142 | pubmed:author | pubmed-author:KimHyo JungHJ | lld:pubmed |
pubmed-article:20164142 | pubmed:author | pubmed-author:WooHye RyunHR | lld:pubmed |
pubmed-article:20164142 | pubmed:author | pubmed-author:LimPyung OkPO | lld:pubmed |
pubmed-article:20164142 | pubmed:author | pubmed-author:LeeIn ChulIC | lld:pubmed |
pubmed-article:20164142 | pubmed:author | pubmed-author:KimJunyoungJ | lld:pubmed |
pubmed-article:20164142 | pubmed:author | pubmed-author:RyuJong... | lld:pubmed |
pubmed-article:20164142 | pubmed:issnType | Electronic | lld:pubmed |
pubmed-article:20164142 | pubmed:volume | 61 | lld:pubmed |
pubmed-article:20164142 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:20164142 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:20164142 | pubmed:pagination | 1419-30 | lld:pubmed |
pubmed-article:20164142 | pubmed:dateRevised | 2010-9-28 | lld:pubmed |
pubmed-article:20164142 | pubmed:meshHeading | pubmed-meshheading:20164142... | lld:pubmed |
pubmed-article:20164142 | pubmed:meshHeading | pubmed-meshheading:20164142... | lld:pubmed |
pubmed-article:20164142 | pubmed:meshHeading | pubmed-meshheading:20164142... | lld:pubmed |
pubmed-article:20164142 | pubmed:meshHeading | pubmed-meshheading:20164142... | lld:pubmed |
pubmed-article:20164142 | pubmed:meshHeading | pubmed-meshheading:20164142... | lld:pubmed |
pubmed-article:20164142 | pubmed:meshHeading | pubmed-meshheading:20164142... | lld:pubmed |
pubmed-article:20164142 | pubmed:meshHeading | pubmed-meshheading:20164142... | lld:pubmed |
pubmed-article:20164142 | pubmed:meshHeading | pubmed-meshheading:20164142... | lld:pubmed |
pubmed-article:20164142 | pubmed:year | 2010 | lld:pubmed |
pubmed-article:20164142 | pubmed:articleTitle | Auxin response factor 2 (ARF2) plays a major role in regulating auxin-mediated leaf longevity. | lld:pubmed |
pubmed-article:20164142 | pubmed:affiliation | Department of Science Education, Jeju National University, 66 Jejudaehakno, Jeju, 690-756, Korea. | lld:pubmed |
pubmed-article:20164142 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:20164142 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
entrez-gene:836321 | entrezgene:pubmed | pubmed-article:20164142 | lld:entrezgene |
http://linkedlifedata.com/r... | entrezgene:pubmed | pubmed-article:20164142 | lld:entrezgene |
http://linkedlifedata.com/r... | pubmed:referesTo | pubmed-article:20164142 | lld:pubmed |
http://linkedlifedata.com/r... | pubmed:referesTo | pubmed-article:20164142 | lld:pubmed |