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pubmed-article:20160437pubmed:abstractTextCurcumin, a polyphenolic compound derived from rhizomes of Curcuma spp., has been shown to possess potent anti-fibrotic properties. Here, we investigate the role of curcumin in modulating the profibrotic action of TGF-beta in human proximal tubule cells (HK-2) and its underlying mechanisms.lld:pubmed
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pubmed-article:20160437pubmed:issn1421-9670lld:pubmed
pubmed-article:20160437pubmed:authorpubmed-author:DuYongYlld:pubmed
pubmed-article:20160437pubmed:authorpubmed-author:WangXuandingXlld:pubmed
pubmed-article:20160437pubmed:authorpubmed-author:ZhuYongliangYlld:pubmed
pubmed-article:20160437pubmed:authorpubmed-author:HuYingYlld:pubmed
pubmed-article:20160437pubmed:authorpubmed-author:HuaLiangLlld:pubmed
pubmed-article:20160437pubmed:copyrightInfo2010 S. Karger AG, Basel.lld:pubmed
pubmed-article:20160437pubmed:issnTypeElectroniclld:pubmed
pubmed-article:20160437pubmed:volume31lld:pubmed
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pubmed-article:20160437pubmed:pagination332-41lld:pubmed
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pubmed-article:20160437pubmed:articleTitleCurcumin inhibits transforming growth factor-beta activity via inhibition of Smad signaling in HK-2 cells.lld:pubmed
pubmed-article:20160437pubmed:affiliationDepartment of Nephrology, Second Affiliated Hospital, Zhe Jiang University School of Medicine, Hangzhou, People's Republic of China.lld:pubmed
pubmed-article:20160437pubmed:publicationTypeJournal Articlelld:pubmed
pubmed-article:20160437pubmed:publicationTypeResearch Support, Non-U.S. Gov'tlld:pubmed