pubmed-article:20150538 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:20150538 | lifeskim:mentions | umls-concept:C0023895 | lld:lifeskim |
pubmed-article:20150538 | lifeskim:mentions | umls-concept:C0035820 | lld:lifeskim |
pubmed-article:20150538 | lifeskim:mentions | umls-concept:C0028754 | lld:lifeskim |
pubmed-article:20150538 | lifeskim:mentions | umls-concept:C0022661 | lld:lifeskim |
pubmed-article:20150538 | lifeskim:mentions | umls-concept:C0403447 | lld:lifeskim |
pubmed-article:20150538 | lifeskim:mentions | umls-concept:C0015695 | lld:lifeskim |
pubmed-article:20150538 | lifeskim:mentions | umls-concept:C0389071 | lld:lifeskim |
pubmed-article:20150538 | lifeskim:mentions | umls-concept:C1150553 | lld:lifeskim |
pubmed-article:20150538 | lifeskim:mentions | umls-concept:C1418898 | lld:lifeskim |
pubmed-article:20150538 | lifeskim:mentions | umls-concept:C1325180 | lld:lifeskim |
pubmed-article:20150538 | lifeskim:mentions | umls-concept:C1325181 | lld:lifeskim |
pubmed-article:20150538 | lifeskim:mentions | umls-concept:C0441712 | lld:lifeskim |
pubmed-article:20150538 | pubmed:issue | 3 | lld:pubmed |
pubmed-article:20150538 | pubmed:dateCreated | 2010-3-1 | lld:pubmed |
pubmed-article:20150538 | pubmed:abstractText | Obesity is a risk factor for chronic kidney disease (CKD) and nonalcoholic fatty liver disease (NAFLD). Recent studies identify mechanisms common to both diseases linked through an interorgan communication orchestrated by fetuin-A and adiponectin. In liver and kidney, the energy sensor 5'-AMP activated protein kinase (AMPK) is pivotal to directing podocytes and hepatocytes to compensatory and potentially deleterious pathways, leading to inflammatory and profibrotic cascades culminating in end-organ damage. Regulation of these early upstream pathways may provide new therapeutic targets for these increasingly common sequelae of obesity. | lld:pubmed |
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pubmed-article:20150538 | pubmed:language | eng | lld:pubmed |
pubmed-article:20150538 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:20150538 | pubmed:citationSubset | IM | lld:pubmed |
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pubmed-article:20150538 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:20150538 | pubmed:month | Mar | lld:pubmed |
pubmed-article:20150538 | pubmed:issn | 1533-3450 | lld:pubmed |
pubmed-article:20150538 | pubmed:author | pubmed-author:SharmaKumarK | lld:pubmed |
pubmed-article:20150538 | pubmed:author | pubmed-author:IxJoachim HJH | lld:pubmed |
pubmed-article:20150538 | pubmed:issnType | Electronic | lld:pubmed |
pubmed-article:20150538 | pubmed:volume | 21 | lld:pubmed |
pubmed-article:20150538 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:20150538 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:20150538 | pubmed:pagination | 406-12 | lld:pubmed |
pubmed-article:20150538 | pubmed:dateRevised | 2011-11-17 | lld:pubmed |
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pubmed-article:20150538 | pubmed:year | 2010 | lld:pubmed |
pubmed-article:20150538 | pubmed:articleTitle | Mechanisms linking obesity, chronic kidney disease, and fatty liver disease: the roles of fetuin-A, adiponectin, and AMPK. | lld:pubmed |
pubmed-article:20150538 | pubmed:affiliation | Division of Nephrology and Hypertension, Department of Medicine, University of California-San Diego/Veterans Affairs San Diego Healthcare System, CA 92093-0711, USA. | lld:pubmed |
pubmed-article:20150538 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:20150538 | pubmed:publicationType | Review | lld:pubmed |
pubmed-article:20150538 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
pubmed-article:20150538 | pubmed:publicationType | Research Support, N.I.H., Extramural | lld:pubmed |
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