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pubmed-article:20149674pubmed:abstractTextPulmonary dysfunction following lung ischaemia-reperfusion is a well-known phenomenon, which may contribute to post-cardiac surgical morbidity. The process is associated with pulmonary inflammatory response and cellular apoptosis. Early molecular mechanisms leading to such lung injury remain largely unknown. We examined whether lung ischaemia and reperfusion cause significant expression changes in numerous genes in the lungs involved in pulmonary apoptosis and other cellular processes by using oligonucleotide microarrays in an experimental model of rodent lung ischaemia-reperfusion injury.lld:pubmed
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pubmed-article:20149674pubmed:authorpubmed-author:RustR CRClld:pubmed
pubmed-article:20149674pubmed:authorpubmed-author:NgCalvin S...lld:pubmed
pubmed-article:20149674pubmed:authorpubmed-author:WanInnes Y...lld:pubmed
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pubmed-article:20149674pubmed:authorpubmed-author:DarziAra WAWlld:pubmed
pubmed-article:20149674pubmed:authorpubmed-author:LauKin MangKMlld:pubmed
pubmed-article:20149674pubmed:authorpubmed-author:UnderwoodMalc...lld:pubmed
pubmed-article:20149674pubmed:copyrightInfoCopyright 2010 European Association for Cardio-Thoracic Surgery. Published by Elsevier B.V. All rights reserved.lld:pubmed
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pubmed-article:20149674pubmed:volume37lld:pubmed
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pubmed-article:20149674pubmed:articleTitleLung ischaemia-reperfusion induced gene expression.lld:pubmed
pubmed-article:20149674pubmed:affiliationDepartment of Surgery, The Chinese University of Hong Kong, Prince of Wales Hospital, Shatin, Hong Kong, China. calvinng@surgery.cuhk.edu.hklld:pubmed
pubmed-article:20149674pubmed:publicationTypeJournal Articlelld:pubmed