| pubmed-article:20126555 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:20126555 | lifeskim:mentions | umls-concept:C0025914 | lld:lifeskim |
| pubmed-article:20126555 | lifeskim:mentions | umls-concept:C0026809 | lld:lifeskim |
| pubmed-article:20126555 | lifeskim:mentions | umls-concept:C0080194 | lld:lifeskim |
| pubmed-article:20126555 | lifeskim:mentions | umls-concept:C0031437 | lld:lifeskim |
| pubmed-article:20126555 | lifeskim:mentions | umls-concept:C1706907 | lld:lifeskim |
| pubmed-article:20126555 | pubmed:issue | 2 | lld:pubmed |
| pubmed-article:20126555 | pubmed:dateCreated | 2010-2-3 | lld:pubmed |
| pubmed-article:20126555 | pubmed:abstractText | Mutations in ATP8B1 (FIC1) underlie cases of cholestatic disease, ranging from chronic and progressive (progressive familial intrahepatic cholestasis) to intermittent (benign recurrent intrahepatic cholestasis). The ATP8B1-deficient mouse serves as an animal model of human ATP8B1 deficiency. | lld:pubmed |
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| pubmed-article:20126555 | pubmed:language | eng | lld:pubmed |
| pubmed-article:20126555 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:20126555 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:20126555 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:20126555 | pubmed:issn | 1932-6203 | lld:pubmed |
| pubmed-article:20126555 | pubmed:author | pubmed-author:ElferinkRonal... | lld:pubmed |
| pubmed-article:20126555 | pubmed:author | pubmed-author:XuHongmeiH | lld:pubmed |
| pubmed-article:20126555 | pubmed:author | pubmed-author:BullLaura NLN | lld:pubmed |
| pubmed-article:20126555 | pubmed:author | pubmed-author:SenSaunakS | lld:pubmed |
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| pubmed-article:20126555 | pubmed:author | pubmed-author:VargasJulie... | lld:pubmed |
| pubmed-article:20126555 | pubmed:author | pubmed-author:GrenertJames... | lld:pubmed |
| pubmed-article:20126555 | pubmed:author | pubmed-author:PaulusmaCoen... | lld:pubmed |
| pubmed-article:20126555 | pubmed:author | pubmed-author:ShahSohelaS | lld:pubmed |
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| pubmed-article:20126555 | pubmed:author | pubmed-author:GroenAnnamiek... | lld:pubmed |
| pubmed-article:20126555 | pubmed:issnType | Electronic | lld:pubmed |
| pubmed-article:20126555 | pubmed:volume | 5 | lld:pubmed |
| pubmed-article:20126555 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:20126555 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:20126555 | pubmed:pagination | e8984 | lld:pubmed |
| pubmed-article:20126555 | pubmed:dateRevised | 2011-6-9 | lld:pubmed |
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| pubmed-article:20126555 | pubmed:year | 2010 | lld:pubmed |
| pubmed-article:20126555 | pubmed:articleTitle | Strain background modifies phenotypes in the ATP8B1-deficient mouse. | lld:pubmed |
| pubmed-article:20126555 | pubmed:affiliation | UCSF Liver Center, University of California San Francisco, San Francisco, California, United States of America. | lld:pubmed |
| pubmed-article:20126555 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:20126555 | pubmed:publicationType | Research Support, N.I.H., Extramural | lld:pubmed |
| entrez-gene:54670 | entrezgene:pubmed | pubmed-article:20126555 | lld:entrezgene |
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