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pubmed-article:2005185pubmed:abstractTextMammalian cells can rapidly make large changes in their rate of polyamine biosynthesis in response to mitogenic and trophic signals. However, cultured cells seem to grow adequately as long as they are supplied a steady but unregulated supply of polyamines. This implies that complex and rapid changes in polyamine synthesis serve a function in a special rather than a general biological context. We suggest that the appropriate context in which regulation of polyamines mediates crucial functions is the mammalian embryo and that one function of polyamines is to act as substrate in an oxidative pathway that arbitrates programmed cell death.lld:pubmed
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pubmed-article:2005185pubmed:dateRevised2007-11-15lld:pubmed
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pubmed-article:2005185pubmed:articleTitleKiller polyamines?lld:pubmed
pubmed-article:2005185pubmed:affiliationDepartment of Microbiology and Immunology, University of California, San Francisco 94143.lld:pubmed
pubmed-article:2005185pubmed:publicationTypeJournal Articlelld:pubmed
pubmed-article:2005185pubmed:publicationTypeResearch Support, U.S. Gov't, P.H.S.lld:pubmed
pubmed-article:2005185pubmed:publicationTypeResearch Support, U.S. Gov't, Non-P.H.S.lld:pubmed
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