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pubmed-article:20031601 | lifeskim:mentions | umls-concept:C0030705 | lld:lifeskim |
pubmed-article:20031601 | lifeskim:mentions | umls-concept:C0007193 | lld:lifeskim |
pubmed-article:20031601 | lifeskim:mentions | umls-concept:C0026882 | lld:lifeskim |
pubmed-article:20031601 | lifeskim:mentions | umls-concept:C0205245 | lld:lifeskim |
pubmed-article:20031601 | lifeskim:mentions | umls-concept:C1420828 | lld:lifeskim |
pubmed-article:20031601 | lifeskim:mentions | umls-concept:C0205396 | lld:lifeskim |
pubmed-article:20031601 | lifeskim:mentions | umls-concept:C0205210 | lld:lifeskim |
pubmed-article:20031601 | lifeskim:mentions | umls-concept:C1880022 | lld:lifeskim |
pubmed-article:20031601 | pubmed:issue | 4 | lld:pubmed |
pubmed-article:20031601 | pubmed:dateCreated | 2009-12-24 | lld:pubmed |
pubmed-article:20031601 | pubmed:abstractText | A key issue for cardiovascular genetic medicine is ascertaining if a putative mutation indeed causes dilated cardiomyopathy (DCM). This is critically important as genetic DCM, usually presenting with advanced, life-threatening disease, may be preventable with early intervention in relatives known to carry the mutation. | lld:pubmed |
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pubmed-article:20031601 | pubmed:language | eng | lld:pubmed |
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pubmed-article:20031601 | pubmed:citationSubset | IM | lld:pubmed |
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pubmed-article:20031601 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:20031601 | pubmed:month | Aug | lld:pubmed |
pubmed-article:20031601 | pubmed:issn | 1942-3268 | lld:pubmed |
pubmed-article:20031601 | pubmed:author | pubmed-author:PotterJames... | lld:pubmed |
pubmed-article:20031601 | pubmed:author | pubmed-author:HershbergerRa... | lld:pubmed |
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pubmed-article:20031601 | pubmed:author | pubmed-author:ParksSharie... | lld:pubmed |
pubmed-article:20031601 | pubmed:author | pubmed-author:LiDuanxiangD | lld:pubmed |
pubmed-article:20031601 | pubmed:author | pubmed-author:ParvatiyarMic... | lld:pubmed |
pubmed-article:20031601 | pubmed:author | pubmed-author:CowanJasonJ | lld:pubmed |
pubmed-article:20031601 | pubmed:author | pubmed-author:PintoJose... | lld:pubmed |
pubmed-article:20031601 | pubmed:issnType | Electronic | lld:pubmed |
pubmed-article:20031601 | pubmed:volume | 2 | lld:pubmed |
pubmed-article:20031601 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:20031601 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:20031601 | pubmed:pagination | 306-13 | lld:pubmed |
pubmed-article:20031601 | pubmed:dateRevised | 2011-4-14 | lld:pubmed |
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pubmed-article:20031601 | pubmed:year | 2009 | lld:pubmed |
pubmed-article:20031601 | pubmed:articleTitle | Clinical and functional characterization of TNNT2 mutations identified in patients with dilated cardiomyopathy. | lld:pubmed |
pubmed-article:20031601 | pubmed:affiliation | Division of Cardiovascular Medicine, Department of Medicine, University of Miami Miller School of Medicine, Miami, FL 33136, USA. rhershberger@med.miami.edu | lld:pubmed |
pubmed-article:20031601 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:20031601 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
pubmed-article:20031601 | pubmed:publicationType | Research Support, N.I.H., Extramural | lld:pubmed |
entrez-gene:7139 | entrezgene:pubmed | pubmed-article:20031601 | lld:entrezgene |
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