| pubmed-article:20009079 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:20009079 | lifeskim:mentions | umls-concept:C0038644 | lld:lifeskim |
| pubmed-article:20009079 | lifeskim:mentions | umls-concept:C0018787 | lld:lifeskim |
| pubmed-article:20009079 | lifeskim:mentions | umls-concept:C0026882 | lld:lifeskim |
| pubmed-article:20009079 | lifeskim:mentions | umls-concept:C0037473 | lld:lifeskim |
| pubmed-article:20009079 | lifeskim:mentions | umls-concept:C0442805 | lld:lifeskim |
| pubmed-article:20009079 | lifeskim:mentions | umls-concept:C0205396 | lld:lifeskim |
| pubmed-article:20009079 | lifeskim:mentions | umls-concept:C0521116 | lld:lifeskim |
| pubmed-article:20009079 | lifeskim:mentions | umls-concept:C1524003 | lld:lifeskim |
| pubmed-article:20009079 | lifeskim:mentions | umls-concept:C0205087 | lld:lifeskim |
| pubmed-article:20009079 | pubmed:issue | 6 | lld:pubmed |
| pubmed-article:20009079 | pubmed:dateCreated | 2009-12-16 | lld:pubmed |
| pubmed-article:20009079 | pubmed:abstractText | Sudden infant death syndrome (SIDS) is a leading cause of death during the first 6 months after birth. About 5% to 10% of SIDS may stem from cardiac channelopathies such as long-QT syndrome. We recently implicated mutations in alpha1-syntrophin (SNTA1) as a novel cause of long-QT syndrome, whereby mutant SNTA1 released inhibition of associated neuronal nitric oxide synthase by the plasma membrane Ca-ATPase PMCA4b, causing increased peak and late sodium current (I(Na)) via S-nitrosylation of the cardiac sodium channel. This study determined the prevalence and functional properties of SIDS-associated SNTA1 mutations. | lld:pubmed |
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| pubmed-article:20009079 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:20009079 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:20009079 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
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| pubmed-article:20009079 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:20009079 | pubmed:month | Dec | lld:pubmed |
| pubmed-article:20009079 | pubmed:issn | 1941-3084 | lld:pubmed |
| pubmed-article:20009079 | pubmed:author | pubmed-author:EdeMM | lld:pubmed |
| pubmed-article:20009079 | pubmed:author | pubmed-author:VattaMatteoM | lld:pubmed |
| pubmed-article:20009079 | pubmed:author | pubmed-author:AckermanMicha... | lld:pubmed |
| pubmed-article:20009079 | pubmed:author | pubmed-author:TesterDavid... | lld:pubmed |
| pubmed-article:20009079 | pubmed:author | pubmed-author:JanuaryCraig... | lld:pubmed |
| pubmed-article:20009079 | pubmed:author | pubmed-author:MakielskiJona... | lld:pubmed |
| pubmed-article:20009079 | pubmed:author | pubmed-author:Medeiros-Domi... | lld:pubmed |
| pubmed-article:20009079 | pubmed:author | pubmed-author:ValdiviaCarme... | lld:pubmed |
| pubmed-article:20009079 | pubmed:author | pubmed-author:Van... | lld:pubmed |
| pubmed-article:20009079 | pubmed:author | pubmed-author:KrobothStacie... | lld:pubmed |
| pubmed-article:20009079 | pubmed:author | pubmed-author:ChengJianding... | lld:pubmed |
| pubmed-article:20009079 | pubmed:author | pubmed-author:TanBi-huaBH | lld:pubmed |
| pubmed-article:20009079 | pubmed:issnType | Electronic | lld:pubmed |
| pubmed-article:20009079 | pubmed:volume | 2 | lld:pubmed |
| pubmed-article:20009079 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:20009079 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:20009079 | pubmed:pagination | 667-76 | lld:pubmed |
| pubmed-article:20009079 | pubmed:dateRevised | 2011-7-22 | lld:pubmed |
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| pubmed-article:20009079 | pubmed:year | 2009 | lld:pubmed |
| pubmed-article:20009079 | pubmed:articleTitle | Alpha1-syntrophin mutations identified in sudden infant death syndrome cause an increase in late cardiac sodium current. | lld:pubmed |
| pubmed-article:20009079 | pubmed:affiliation | Division of Cardiovascular Medicine, Department of Medicine, University of Wisconsin, Madison, WI, USA. | lld:pubmed |
| pubmed-article:20009079 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:20009079 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
| pubmed-article:20009079 | pubmed:publicationType | Research Support, N.I.H., Extramural | lld:pubmed |
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