| pubmed-article:1989582 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:1989582 | lifeskim:mentions | umls-concept:C0205103 | lld:lifeskim |
| pubmed-article:1989582 | lifeskim:mentions | umls-concept:C0026237 | lld:lifeskim |
| pubmed-article:1989582 | lifeskim:mentions | umls-concept:C0752063 | lld:lifeskim |
| pubmed-article:1989582 | lifeskim:mentions | umls-concept:C1882726 | lld:lifeskim |
| pubmed-article:1989582 | lifeskim:mentions | umls-concept:C1514468 | lld:lifeskim |
| pubmed-article:1989582 | pubmed:dateCreated | 1991-2-27 | lld:pubmed |
| pubmed-article:1989582 | pubmed:abstractText | 1. The synthesis of [U-14C]hexadecanedionoyl-mono-CoA is described. 2. The beta-oxidation of [U-14C]hexadecanedionoyl-mono-CoA by purified rat liver peroxisomes and mitochondria is demonstrated. 3. The products of mitochondrial beta-oxidation of [U-14C]hexadecanedionoyl-mono-CoA include ketone bodies, citrate and acetylcarnitine. 4. Tetradecadionoyl-mono-CoA, hexadec-2-enedionyl-mono-CoA and hexadionoyl-mono-CoA were the only detectable intermediates formed by mitochondrial beta-oxidation, whereas acetyl-CoA and all saturated even-numbered intermediates of chain length C6-C16 were generated by peroxisomal beta-oxidation. 5. Hexadecanedionoyl-mono-CoA and hexadecanoyl-CoA were equally effective substrates for peroxisomal beta-oxidation, but hexadecanedionoyl-mono-CoA was a relatively poorer substrate for the mitochondrial pathway. | lld:pubmed |
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| pubmed-article:1989582 | pubmed:language | eng | lld:pubmed |
| pubmed-article:1989582 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:1989582 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:1989582 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:1989582 | pubmed:month | Jan | lld:pubmed |
| pubmed-article:1989582 | pubmed:issn | 0264-6021 | lld:pubmed |
| pubmed-article:1989582 | pubmed:author | pubmed-author:BartlettKK | lld:pubmed |
| pubmed-article:1989582 | pubmed:author | pubmed-author:PourfarzamMM | lld:pubmed |
| pubmed-article:1989582 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:1989582 | pubmed:day | 1 | lld:pubmed |
| pubmed-article:1989582 | pubmed:volume | 273(Pt 1) | lld:pubmed |
| pubmed-article:1989582 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:1989582 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:1989582 | pubmed:pagination | 205-10 | lld:pubmed |
| pubmed-article:1989582 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
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| pubmed-article:1989582 | pubmed:year | 1991 | lld:pubmed |
| pubmed-article:1989582 | pubmed:articleTitle | Products and intermediates of the beta-oxidation of [U-14C]hexadecanedionoyl-mono-CoA by rat liver peroxisomes and mitochondria. | lld:pubmed |
| pubmed-article:1989582 | pubmed:affiliation | Department of Child Health, Medical School, University of Newcastle upon Tyne, U.K. | lld:pubmed |
| pubmed-article:1989582 | pubmed:publicationType | Journal Article | lld:pubmed |
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