| pubmed-article:19849853 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:19849853 | lifeskim:mentions | umls-concept:C1963704 | lld:lifeskim |
| pubmed-article:19849853 | lifeskim:mentions | umls-concept:C0242606 | lld:lifeskim |
| pubmed-article:19849853 | lifeskim:mentions | umls-concept:C0277785 | lld:lifeskim |
| pubmed-article:19849853 | lifeskim:mentions | umls-concept:C0851346 | lld:lifeskim |
| pubmed-article:19849853 | lifeskim:mentions | umls-concept:C0596263 | lld:lifeskim |
| pubmed-article:19849853 | lifeskim:mentions | umls-concept:C0205234 | lld:lifeskim |
| pubmed-article:19849853 | lifeskim:mentions | umls-concept:C1136359 | lld:lifeskim |
| pubmed-article:19849853 | pubmed:dateCreated | 2009-11-12 | lld:pubmed |
| pubmed-article:19849853 | pubmed:abstractText | Hazardous health effects stemming from exposure to radiofrequency electromagnetic waves (RF-EMW) emitted from cell phones have been reported in the literature. However, the cellular target of RF-EMW is still controversial. This review identifies the plasma membrane as a target of RF-EMW. In addition, the effects of RF-EMW on plasma membrane structures (i.e. NADH oxidase, phosphatidylserine, ornithine decarboxylase) and voltage-gated calcium channels are discussed. We explore the disturbance in reactive oxygen species (ROS) metabolism caused by RF-EMW and delineate NADH oxidase mediated ROS formation as playing a central role in oxidative stress (OS) due to cell phone radiation (with a focus on the male reproductive system). This review also addresses: 1) the controversial effects of RF-EMW on mammalian cells and sperm DNA as well as its effect on apoptosis, 2) epidemiological, in vivo animal and in vitro studies on the effect of RF-EMW on male reproductive system, and 3) finally, exposure assessment and dosimetry by computational biomodeling. | lld:pubmed |
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