pubmed-article:19848064 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:19848064 | lifeskim:mentions | umls-concept:C1257890 | lld:lifeskim |
pubmed-article:19848064 | lifeskim:mentions | umls-concept:C0281361 | lld:lifeskim |
pubmed-article:19848064 | lifeskim:mentions | umls-concept:C0005516 | lld:lifeskim |
pubmed-article:19848064 | lifeskim:mentions | umls-concept:C0425245 | lld:lifeskim |
pubmed-article:19848064 | lifeskim:mentions | umls-concept:C0302350 | lld:lifeskim |
pubmed-article:19848064 | lifeskim:mentions | umls-concept:C0679622 | lld:lifeskim |
pubmed-article:19848064 | lifeskim:mentions | umls-concept:C0205314 | lld:lifeskim |
pubmed-article:19848064 | lifeskim:mentions | umls-concept:C1521840 | lld:lifeskim |
pubmed-article:19848064 | pubmed:issue | 5 | lld:pubmed |
pubmed-article:19848064 | pubmed:dateCreated | 2009-10-23 | lld:pubmed |
pubmed-article:19848064 | pubmed:abstractText | High mobility group A1 (HMGA1) proteins are architectural transcriptional factors that are over-expressed in a wide range of human malignancies. Recently published evidence suggests HMGA1 is a promising candidate biomarker and therapeutic target in pancreatic cancer. This review summarises data implicating HMGA1 as an important mediator of progression in human cancer and in pancreatic cancer. | lld:pubmed |
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