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| pubmed-article:19815821 | pubmed:issue | 11 | lld:pubmed |
| pubmed-article:19815821 | pubmed:dateCreated | 2009-11-20 | lld:pubmed |
| pubmed-article:19815821 | pubmed:abstractText | Aldehyde accumulation is regarded as a pathognomonic feature of oxidative stress-associated cardiovascular disease. | lld:pubmed |
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| pubmed-article:19815821 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:19815821 | pubmed:month | Nov | lld:pubmed |
| pubmed-article:19815821 | pubmed:issn | 1524-4571 | lld:pubmed |
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| pubmed-article:19815821 | pubmed:volume | 105 | lld:pubmed |
| pubmed-article:19815821 | pubmed:owner | NLM | lld:pubmed |
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| pubmed-article:19815821 | pubmed:year | 2009 | lld:pubmed |
| pubmed-article:19815821 | pubmed:articleTitle | Metabolic remodeling induced by mitochondrial aldehyde stress stimulates tolerance to oxidative stress in the heart. | lld:pubmed |
| pubmed-article:19815821 | pubmed:affiliation | Department of Regenerative Medicine and Advanced Cardiac Therapeutics, Keio University School of Medicine, 35 Shinanomachi Shinjuku-ku, Tokyo 160-8582, Japan. | lld:pubmed |
| pubmed-article:19815821 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:19815821 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
| pubmed-article:19815821 | pubmed:publicationType | Research Support, N.I.H., Extramural | lld:pubmed |
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