| pubmed-article:19747917 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:19747917 | lifeskim:mentions | umls-concept:C0022688 | lld:lifeskim |
| pubmed-article:19747917 | lifeskim:mentions | umls-concept:C0596402 | lld:lifeskim |
| pubmed-article:19747917 | lifeskim:mentions | umls-concept:C0524910 | lld:lifeskim |
| pubmed-article:19747917 | pubmed:issue | 1 | lld:pubmed |
| pubmed-article:19747917 | pubmed:dateCreated | 2010-1-28 | lld:pubmed |
| pubmed-article:19747917 | pubmed:abstractText | Patients with chronic hepatitis C virus (HCV) infection display great variability in disease activity and progression. Although virus-specific adaptive immune responses have been characterized extensively and found to be impaired in chronic hepatitis C, the role of innate immune responses in disease activity and progression of chronic hepatitis C is not well understood. | lld:pubmed |
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| pubmed-article:19747917 | pubmed:language | eng | lld:pubmed |
| pubmed-article:19747917 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:19747917 | pubmed:citationSubset | AIM | lld:pubmed |
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| pubmed-article:19747917 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:19747917 | pubmed:month | Jan | lld:pubmed |
| pubmed-article:19747917 | pubmed:issn | 1528-0012 | lld:pubmed |
| pubmed-article:19747917 | pubmed:author | pubmed-author:HoofnagleJay... | lld:pubmed |
| pubmed-article:19747917 | pubmed:author | pubmed-author:LiangT JakeTJ | lld:pubmed |
| pubmed-article:19747917 | pubmed:author | pubmed-author:RehermannBarb... | lld:pubmed |
| pubmed-article:19747917 | pubmed:author | pubmed-author:AhlenstielGol... | lld:pubmed |
| pubmed-article:19747917 | pubmed:author | pubmed-author:HellerTheoT | lld:pubmed |
| pubmed-article:19747917 | pubmed:author | pubmed-author:RotmanYaronY | lld:pubmed |
| pubmed-article:19747917 | pubmed:author | pubmed-author:GhanyMarc GMG | lld:pubmed |
| pubmed-article:19747917 | pubmed:author | pubmed-author:FeldJordan... | lld:pubmed |
| pubmed-article:19747917 | pubmed:author | pubmed-author:KohChristophe... | lld:pubmed |
| pubmed-article:19747917 | pubmed:author | pubmed-author:TiterenceRach... | lld:pubmed |
| pubmed-article:19747917 | pubmed:author | pubmed-author:EdlichBirgitB | lld:pubmed |
| pubmed-article:19747917 | pubmed:copyrightInfo | Copyright 2010 AGA Institute. Published by Elsevier Inc. All rights reserved. | lld:pubmed |
| pubmed-article:19747917 | pubmed:issnType | Electronic | lld:pubmed |
| pubmed-article:19747917 | pubmed:volume | 138 | lld:pubmed |
| pubmed-article:19747917 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:19747917 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:19747917 | pubmed:pagination | 325-35.e1-2 | lld:pubmed |
| pubmed-article:19747917 | pubmed:dateRevised | 2011-7-19 | lld:pubmed |
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| pubmed-article:19747917 | pubmed:year | 2010 | lld:pubmed |
| pubmed-article:19747917 | pubmed:articleTitle | Natural killer cells are polarized toward cytotoxicity in chronic hepatitis C in an interferon-alfa-dependent manner. | lld:pubmed |
