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pubmed-article:19729606pubmed:abstractTextLocalization of atherosclerotic plaques typically correlates with areas of biomechanical strain where shear stress is decreased while stretch, thought to promote atherogenesis through enhanced oxidative stress, is increased.lld:pubmed
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pubmed-article:19729606pubmed:articleTitleUpregulation of glutathione peroxidase offsets stretch-induced proatherogenic gene expression in human endothelial cells.lld:pubmed
pubmed-article:19729606pubmed:affiliationInstitute of Physiology and Pathophysiology, Division of Cardiovascular Physiology, University Hospital Heidelberg, Heidelberg, Germany.lld:pubmed
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