pubmed-article:19620505 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:19620505 | lifeskim:mentions | umls-concept:C0178324 | lld:lifeskim |
pubmed-article:19620505 | lifeskim:mentions | umls-concept:C1704632 | lld:lifeskim |
pubmed-article:19620505 | lifeskim:mentions | umls-concept:C0871261 | lld:lifeskim |
pubmed-article:19620505 | lifeskim:mentions | umls-concept:C2911692 | lld:lifeskim |
pubmed-article:19620505 | lifeskim:mentions | umls-concept:C1706817 | lld:lifeskim |
pubmed-article:19620505 | lifeskim:mentions | umls-concept:C0205460 | lld:lifeskim |
pubmed-article:19620505 | lifeskim:mentions | umls-concept:C1511545 | lld:lifeskim |
pubmed-article:19620505 | pubmed:issue | 5 | lld:pubmed |
pubmed-article:19620505 | pubmed:dateCreated | 2009-8-4 | lld:pubmed |
pubmed-article:19620505 | pubmed:abstractText | Myeloid-related protein (MRP)-8 (S100A8) and MRP-14 (S100A9) are members of the S100 family of calcium-modulated proteins that regulate myeloid cell function and control inflammation, in part, through activation of Toll-like receptor-4 and the receptor for advanced glycation end products. A transcriptional profiling approach in patients with acute coronary syndromes identified MRP-14 as a novel predictor of myocardial infarction. Further studies demonstrated that elevated plasma levels of MRP-8/14 heterodimer predict increased risk of first and recurrent cardiovascular events. Beyond its serving as a risk marker, whether MRP-8/14 participates directly in vascular inflammation and disease remains unclear. | lld:pubmed |
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pubmed-article:19620505 | pubmed:language | eng | lld:pubmed |
pubmed-article:19620505 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:19620505 | pubmed:citationSubset | AIM | lld:pubmed |
pubmed-article:19620505 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
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pubmed-article:19620505 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:19620505 | pubmed:month | Aug | lld:pubmed |
pubmed-article:19620505 | pubmed:issn | 1524-4539 | lld:pubmed |
pubmed-article:19620505 | pubmed:author | pubmed-author:ZhuK XKX | lld:pubmed |
pubmed-article:19620505 | pubmed:author | pubmed-author:HoggNancyN | lld:pubmed |
pubmed-article:19620505 | pubmed:author | pubmed-author:SukhovaGalina... | lld:pubmed |
pubmed-article:19620505 | pubmed:author | pubmed-author:LibbyPeterP | lld:pubmed |
pubmed-article:19620505 | pubmed:author | pubmed-author:SakumaMasashi... | lld:pubmed |
pubmed-article:19620505 | pubmed:author | pubmed-author:SimonDaniel... | lld:pubmed |
pubmed-article:19620505 | pubmed:author | pubmed-author:CroceKevinK | lld:pubmed |
pubmed-article:19620505 | pubmed:author | pubmed-author:WangYunmeiY | lld:pubmed |
pubmed-article:19620505 | pubmed:author | pubmed-author:PackardRené... | lld:pubmed |
pubmed-article:19620505 | pubmed:author | pubmed-author:GaoHuiyunH | lld:pubmed |
pubmed-article:19620505 | pubmed:author | pubmed-author:MoorokaToshif... | lld:pubmed |
pubmed-article:19620505 | pubmed:issnType | Electronic | lld:pubmed |
pubmed-article:19620505 | pubmed:day | 4 | lld:pubmed |
pubmed-article:19620505 | pubmed:volume | 120 | lld:pubmed |
pubmed-article:19620505 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:19620505 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:19620505 | pubmed:pagination | 427-36 | lld:pubmed |
pubmed-article:19620505 | pubmed:dateRevised | 2011-7-27 | lld:pubmed |
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