pubmed-article:19590993 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:19590993 | lifeskim:mentions | umls-concept:C0087111 | lld:lifeskim |
pubmed-article:19590993 | lifeskim:mentions | umls-concept:C0027726 | lld:lifeskim |
pubmed-article:19590993 | lifeskim:mentions | umls-concept:C0393022 | lld:lifeskim |
pubmed-article:19590993 | lifeskim:mentions | umls-concept:C0002726 | lld:lifeskim |
pubmed-article:19590993 | lifeskim:mentions | umls-concept:C0678226 | lld:lifeskim |
pubmed-article:19590993 | pubmed:issue | 3 | lld:pubmed |
pubmed-article:19590993 | pubmed:dateCreated | 2010-6-11 | lld:pubmed |
pubmed-article:19590993 | pubmed:abstractText | We report a patient with AH amyloidosis associated with lymphoplasmacytic leukemia that has remained in a stable state with a nephrotic syndrome for 17 months since the commencement of cyclic rituximab therapy aimed at suppression of pathogenetic gamma heavy chains. Free light chains in serum and CD20-positive cells in peripheral blood were useful as hematological markers in the patient. Rituximab might be a potent therapeutic option for AH amyloidosis associated with a B-cell lymphoproliferative disorder. | lld:pubmed |
pubmed-article:19590993 | pubmed:language | eng | lld:pubmed |
pubmed-article:19590993 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:19590993 | pubmed:citationSubset | IM | lld:pubmed |
pubmed-article:19590993 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:19590993 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:19590993 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:19590993 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:19590993 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:19590993 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:19590993 | pubmed:issn | 1744-2818 | lld:pubmed |
pubmed-article:19590993 | pubmed:author | pubmed-author:MatsudaMasayu... | lld:pubmed |
pubmed-article:19590993 | pubmed:author | pubmed-author:YazakiMasahid... | lld:pubmed |
pubmed-article:19590993 | pubmed:author | pubmed-author:GonoTakahisaT | lld:pubmed |
pubmed-article:19590993 | pubmed:author | pubmed-author:IkedaShu-Ichi... | lld:pubmed |
pubmed-article:19590993 | pubmed:author | pubmed-author:KatohNagaakiN | lld:pubmed |
pubmed-article:19590993 | pubmed:author | pubmed-author:MiyazakiDaigo... | lld:pubmed |
pubmed-article:19590993 | pubmed:issnType | Electronic | lld:pubmed |
pubmed-article:19590993 | pubmed:volume | 16 | lld:pubmed |
pubmed-article:19590993 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:19590993 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:19590993 | pubmed:pagination | 178-80 | lld:pubmed |
pubmed-article:19590993 | pubmed:dateRevised | 2010-11-18 | lld:pubmed |
pubmed-article:19590993 | pubmed:meshHeading | pubmed-meshheading:19590993... | lld:pubmed |
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pubmed-article:19590993 | pubmed:meshHeading | pubmed-meshheading:19590993... | lld:pubmed |
pubmed-article:19590993 | pubmed:year | 2009 | lld:pubmed |
pubmed-article:19590993 | pubmed:articleTitle | Rituximab therapy in nephrotic syndrome due to AH amyloidosis. | lld:pubmed |
pubmed-article:19590993 | pubmed:publicationType | Letter | lld:pubmed |
pubmed-article:19590993 | pubmed:publicationType | Case Reports | lld:pubmed |
pubmed-article:19590993 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |