| pubmed-article:19590240 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:19590240 | lifeskim:mentions | umls-concept:C0026336 | lld:lifeskim |
| pubmed-article:19590240 | lifeskim:mentions | umls-concept:C0022646 | lld:lifeskim |
| pubmed-article:19590240 | lifeskim:mentions | umls-concept:C0538674 | lld:lifeskim |
| pubmed-article:19590240 | lifeskim:mentions | umls-concept:C0018988 | lld:lifeskim |
| pubmed-article:19590240 | lifeskim:mentions | umls-concept:C0449258 | lld:lifeskim |
| pubmed-article:19590240 | lifeskim:mentions | umls-concept:C0599946 | lld:lifeskim |
| pubmed-article:19590240 | pubmed:issue | 1 | lld:pubmed |
| pubmed-article:19590240 | pubmed:dateCreated | 2009-8-26 | lld:pubmed |
| pubmed-article:19590240 | pubmed:abstractText | Heme oxygenase-1 (HO-1) has been shown to protect against fibrotic proliferation and apoptosis in several models of renal damage. The purpose of this study was to evaluate the impact of a treatment with the HO-1 inducer hemin on the progression of chronic kidney disease in nephrectomized rats versus the AT1 receptor antagonist losartan. | lld:pubmed |
| pubmed-article:19590240 | pubmed:language | eng | lld:pubmed |
| pubmed-article:19590240 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:19590240 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:19590240 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:19590240 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:19590240 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:19590240 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:19590240 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:19590240 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:19590240 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:19590240 | pubmed:issn | 1660-2129 | lld:pubmed |
| pubmed-article:19590240 | pubmed:author | pubmed-author:MachetMarie-C... | lld:pubmed |
| pubmed-article:19590240 | pubmed:author | pubmed-author:HalimiJean-Mi... | lld:pubmed |
| pubmed-article:19590240 | pubmed:author | pubmed-author:GarrigueMarie... | lld:pubmed |
| pubmed-article:19590240 | pubmed:author | pubmed-author:HyvelinJean-M... | lld:pubmed |
| pubmed-article:19590240 | pubmed:author | pubmed-author:AntierDanielD | lld:pubmed |
| pubmed-article:19590240 | pubmed:author | pubmed-author:EderVéronique... | lld:pubmed |
| pubmed-article:19590240 | pubmed:author | pubmed-author:DesbuardsNico... | lld:pubmed |
| pubmed-article:19590240 | pubmed:copyrightInfo | Copyright 2009 S. Karger AG, Basel. | lld:pubmed |
| pubmed-article:19590240 | pubmed:issnType | Electronic | lld:pubmed |
| pubmed-article:19590240 | pubmed:volume | 113 | lld:pubmed |
| pubmed-article:19590240 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:19590240 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:19590240 | pubmed:pagination | e35-44 | lld:pubmed |
| pubmed-article:19590240 | pubmed:meshHeading | pubmed-meshheading:19590240... | lld:pubmed |
| pubmed-article:19590240 | pubmed:meshHeading | pubmed-meshheading:19590240... | lld:pubmed |
| pubmed-article:19590240 | pubmed:meshHeading | pubmed-meshheading:19590240... | lld:pubmed |
| pubmed-article:19590240 | pubmed:meshHeading | pubmed-meshheading:19590240... | lld:pubmed |
| pubmed-article:19590240 | pubmed:meshHeading | pubmed-meshheading:19590240... | lld:pubmed |
| pubmed-article:19590240 | pubmed:meshHeading | pubmed-meshheading:19590240... | lld:pubmed |
| pubmed-article:19590240 | pubmed:meshHeading | pubmed-meshheading:19590240... | lld:pubmed |
| pubmed-article:19590240 | pubmed:meshHeading | pubmed-meshheading:19590240... | lld:pubmed |
| pubmed-article:19590240 | pubmed:meshHeading | pubmed-meshheading:19590240... | lld:pubmed |
| pubmed-article:19590240 | pubmed:meshHeading | pubmed-meshheading:19590240... | lld:pubmed |
| pubmed-article:19590240 | pubmed:meshHeading | pubmed-meshheading:19590240... | lld:pubmed |
| pubmed-article:19590240 | pubmed:meshHeading | pubmed-meshheading:19590240... | lld:pubmed |
| pubmed-article:19590240 | pubmed:meshHeading | pubmed-meshheading:19590240... | lld:pubmed |
| pubmed-article:19590240 | pubmed:meshHeading | pubmed-meshheading:19590240... | lld:pubmed |
| pubmed-article:19590240 | pubmed:meshHeading | pubmed-meshheading:19590240... | lld:pubmed |
| pubmed-article:19590240 | pubmed:meshHeading | pubmed-meshheading:19590240... | lld:pubmed |
| pubmed-article:19590240 | pubmed:meshHeading | pubmed-meshheading:19590240... | lld:pubmed |
| pubmed-article:19590240 | pubmed:year | 2009 | lld:pubmed |
| pubmed-article:19590240 | pubmed:articleTitle | Heme oxygenase-1 inducer hemin attenuates the progression of remnant kidney model. | lld:pubmed |
| pubmed-article:19590240 | pubmed:affiliation | LABPART, EA 3852, IFR135, Université François Rabelais, FR-37032 Tours, France. | lld:pubmed |
| pubmed-article:19590240 | pubmed:publicationType | Journal Article | lld:pubmed |
