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pubmed-article:19582162pubmed:abstractTextMouse AA-amyloidosis is a transmissible disease by a prion-like mechanism where amyloid fibrils act by seeding. Synthetic peptides with no amyloid relationship can assemble into amyloid-like fibrils and these may have seeding capacity for amyloid proteins.lld:pubmed
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pubmed-article:19582162pubmed:articleTitleFibrils from designed non-amyloid-related synthetic peptides induce AA-amyloidosis during inflammation in an animal model.lld:pubmed
pubmed-article:19582162pubmed:affiliationRudbeck Laboratory, Department of Genetics and Pathology, Uppsala University, Uppsala, Sweden.lld:pubmed
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