| pubmed-article:19555469 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:19555469 | lifeskim:mentions | umls-concept:C0003873 | lld:lifeskim |
| pubmed-article:19555469 | lifeskim:mentions | umls-concept:C0035647 | lld:lifeskim |
| pubmed-article:19555469 | lifeskim:mentions | umls-concept:C0017337 | lld:lifeskim |
| pubmed-article:19555469 | lifeskim:mentions | umls-concept:C0033522 | lld:lifeskim |
| pubmed-article:19555469 | lifeskim:mentions | umls-concept:C0919646 | lld:lifeskim |
| pubmed-article:19555469 | pubmed:issue | 3 | lld:pubmed |
| pubmed-article:19555469 | pubmed:dateCreated | 2009-7-22 | lld:pubmed |
| pubmed-article:19555469 | pubmed:abstractText | Rheumatoid arthritis (RA) is more common in females than males and sex steroid hormones may in part explain this difference. We conducted a case-control study nested within two prospective studies to determine the associations between plasma steroid hormones measured prior to RA onset and polymorphisms in the androgen receptor (AR), estrogen receptor 2 (ESR2), aromatase (CYP19) and progesterone receptor (PGR) genes and RA risk. | lld:pubmed |
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| pubmed-article:19555469 | pubmed:language | eng | lld:pubmed |
| pubmed-article:19555469 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:19555469 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:19555469 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:19555469 | pubmed:issn | 1478-6362 | lld:pubmed |
| pubmed-article:19555469 | pubmed:author | pubmed-author:HankinsonSusa... | lld:pubmed |
| pubmed-article:19555469 | pubmed:author | pubmed-author:LeeI-MinIM | lld:pubmed |
| pubmed-article:19555469 | pubmed:author | pubmed-author:KarlsonElizab... | lld:pubmed |
| pubmed-article:19555469 | pubmed:author | pubmed-author:De... | lld:pubmed |
| pubmed-article:19555469 | pubmed:author | pubmed-author:BuringJulieJ | lld:pubmed |
| pubmed-article:19555469 | pubmed:author | pubmed-author:FraserPatrici... | lld:pubmed |
